The L-type calcium channel C-terminus: sparking interest beyond its role in calcium-dependent inactivation.
作者信息
Zamponi Gerald W
机构信息
Department of Physiology and Biophysics, Cellular and Molecular Neurobiology Research Group, University of Calgary, Calgary T2N 4N1, Canada.
出版信息
J Physiol. 2003 Oct 15;552(Pt 2):333. doi: 10.1113/jphysiol.2003.052852.
Abstract
摘要
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Engineered calmodulins reveal the unexpected eminence of Ca2+ channel inactivation in controlling heart excitation.工程改造的钙调蛋白揭示了钙离子通道失活在控制心脏兴奋方面出人意料的重要性。
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Calmodulin priming: nuclear translocation of a calmodulin complex and the memory of prior neuronal activity.钙调蛋白引发：钙调蛋白复合物的核转位及对先前神经元活动的记忆
Proc Natl Acad Sci U S A. 2001 Dec 18;98(26):15342-7. doi: 10.1073/pnas.211563998. Epub 2001 Dec 11.
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Signaling to the nucleus by an L-type calcium channel-calmodulin complex through the MAP kinase pathway.L型钙通道-钙调蛋白复合物通过丝裂原活化蛋白激酶途径向细胞核发出信号。
Science. 2001 Oct 12;294(5541):333-9. doi: 10.1126/science.1063395.
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Cardiac excitation-contraction coupling: role of membrane potential in regulation of contraction.心脏兴奋-收缩偶联：膜电位在收缩调节中的作用。
Am J Physiol Heart Circ Physiol. 2001 May;280(5):H1928-44. doi: 10.1152/ajpheart.2001.280.5.H1928.
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Calmodulin is the Ca2+ sensor for Ca2+ -dependent inactivation of L-type calcium channels.钙调蛋白是L型钙通道钙依赖性失活的钙离子传感器。
Neuron. 1999 Mar;22(3):549-58. doi: 10.1016/s0896-6273(00)80709-6.
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Enhanced dihydropyridine receptor channel activity in the presence of ryanodine receptor.在存在兰尼碱受体的情况下增强的二氢吡啶受体通道活性。
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Calcium sparks: elementary events underlying excitation-contraction coupling in heart muscle.钙火花：心肌兴奋-收缩偶联的基本事件。
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Regions of the skeletal muscle dihydropyridine receptor critical for excitation-contraction coupling.骨骼肌二氢吡啶受体中对兴奋-收缩偶联至关重要的区域。
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