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反射性癫痫与非酮症性高血糖症。

Reflex epilepsy and non-ketotic hyperglycemia.

作者信息

Ozer Feriha, Mutlu Aytul, Ozkayran Tufan

机构信息

Haseki Education and Research Hospital, Neurology Clinic, Istanbul, Turkey.

出版信息

Epileptic Disord. 2003 Sep;5(3):165-8.

PMID:14684353
Abstract

Epileptic seizures are paroxysmal events, and it is likely that many, if not most, of them are precipitated by known or unknown factors acting on a central nervous system that is predisposed to the production of epileptic discharges by the presence of an organic lesion, a genetically determined neuronal hyperexcitability, or both. Known precipitating influences are quite varied. We report the case of a 58 years-old patient, followed for non-ketotic hyperglycemia, who presented with focal seizures exclusively induced by specific, active or passive, postures and movements of the right arm or hand. MRI was normal. Following regulation of glucose blood levels the position-induced seizures stopped and antiepileptics were not prescribed. The seizures are kept under control by regulating blood glucose.

摘要

癫痫发作是阵发性事件,很可能许多(即便不是大多数)发作是由作用于中枢神经系统的已知或未知因素诱发的,该中枢神经系统因存在器质性病变、基因决定的神经元兴奋性过高或两者皆有而易于产生癫痫放电。已知的诱发影响多种多样。我们报告一例58岁因非酮症高血糖症接受随访的患者,该患者仅由右臂或右手特定的主动或被动姿势及运动诱发局灶性癫痫发作。磁共振成像(MRI)正常。在血糖水平得到控制后,体位诱发的癫痫发作停止,未开具抗癫痫药物。通过调节血糖,癫痫发作得以控制。

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