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Bortezomib therapy for myeloma.

作者信息

Anderson Kenneth C

出版信息

Curr Hematol Rep. 2004 Jan;3(1):65.

PMID:14695852
Abstract
摘要

相似文献

1
Bortezomib therapy for myeloma.硼替佐米治疗骨髓瘤。
Curr Hematol Rep. 2004 Jan;3(1):65.
2
Bortezomib: a valuable new antineoplastic strategy in multiple myeloma.硼替佐米:多发性骨髓瘤中一种有价值的新型抗肿瘤策略。
Acta Oncol. 2005;44(5):440-8. doi: 10.1080/02841860510030002.
3
Clinical impact of bortezomib in frontline regimens for patients with multiple myeloma.硼替佐米在多发性骨髓瘤患者一线治疗方案中的临床影响。
Oncologist. 2007 Aug;12(8):978-90. doi: 10.1634/theoncologist.12-8-978.
4
Safety of prolonged therapy with bortezomib in relapsed or refractory multiple myeloma.硼替佐米用于复发或难治性多发性骨髓瘤的长期治疗安全性
Cancer. 2005 Nov 15;104(10):2141-8. doi: 10.1002/cncr.21427.
5
Expanding role of bortezomib in multiple myeloma: nursing implications.硼替佐米在多发性骨髓瘤中不断扩大的作用:护理启示
Cancer Nurs. 2008 May-Jun;31(3):239-49. doi: 10.1097/01.NCC.0000305733.80592.8e.
6
Beyond single-agent bortezomib: combination regimens in relapsed multiple myeloma.超越单药硼替佐米:复发多发性骨髓瘤的联合治疗方案
Curr Opin Oncol. 2006 Nov;18(6):598-608. doi: 10.1097/01.cco.0000245320.34658.bd.
7
[Treatment with a proteasome inhibitor, bortezomib, for thalidomide-resistant multiple myeloma].用蛋白酶体抑制剂硼替佐米治疗沙利度胺耐药的多发性骨髓瘤
Rinsho Ketsueki. 2004 Feb;45(2):144-8.
8
Utility of bortezomib retreatment in relapsed or refractory multiple myeloma patients: a multicenter case series.硼替佐米再次治疗复发或难治性多发性骨髓瘤患者的效用:一项多中心病例系列研究
Clin Adv Hematol Oncol. 2008 Oct;6(10):755-60.
9
Neurotoxicity of bortezomib therapy in multiple myeloma: a single-center experience and review of the literature.硼替佐米治疗多发性骨髓瘤的神经毒性:单中心经验及文献综述
Cancer. 2007 Sep 1;110(5):1042-9. doi: 10.1002/cncr.22921.
10
Bortezomib and its role in the management of patients with multiple myeloma.硼替佐米及其在多发性骨髓瘤患者治疗中的作用。
Expert Rev Anticancer Ther. 2004 Apr;4(2):171-9. doi: 10.1586/14737140.4.2.171.

引用本文的文献

1
The fatty acid elongase ELOVL6 regulates bortezomib resistance in multiple myeloma.脂肪酸延长酶 ELOVL6 调控多发性骨髓瘤对硼替佐米的耐药性。
Blood Adv. 2021 Apr 13;5(7):1933-1946. doi: 10.1182/bloodadvances.2020002578.
2
Isoginkgetin, a Natural Biflavonoid Proteasome Inhibitor, Sensitizes Cancer Cells to Apoptosis via Disruption of Lysosomal Homeostasis and Impaired Protein Clearance.异甘草素,一种天然双黄酮蛋白酶体抑制剂,通过破坏溶酶体稳态和损害蛋白清除来诱导癌细胞凋亡。
Mol Cell Biol. 2019 Apr 30;39(10). doi: 10.1128/MCB.00489-18. Print 2019 May 15.
3
Low-dose bortezomib increases the expression of NKG2D and DNAM-1 ligands and enhances induced NK and γδ T cell-mediated lysis in multiple myeloma.
低剂量硼替佐米增加NKG2D和DNAM-1配体的表达,并增强多发性骨髓瘤中诱导的自然杀伤细胞和γδT细胞介导的细胞溶解作用。
Oncotarget. 2017 Jan 24;8(4):5954-5964. doi: 10.18632/oncotarget.13979.
4
Mitochondrial thioredoxin reductase regulates major cytotoxicity pathways of proteasome inhibitors in multiple myeloma cells.线粒体硫氧还蛋白还原酶调节蛋白酶体抑制剂在多发性骨髓瘤细胞中的主要细胞毒性途径。
Leukemia. 2016 Jan;30(1):104-11. doi: 10.1038/leu.2015.190. Epub 2015 Jul 24.
5
Down-regulation of CD9 by methylation decreased bortezomib sensitivity in multiple myeloma.CD9的甲基化下调降低了多发性骨髓瘤对硼替佐米的敏感性。
PLoS One. 2014 May 2;9(5):e95765. doi: 10.1371/journal.pone.0095765. eCollection 2014.
6
Differential downregulation of telomerase activity by bortezomib in multiple myeloma cells-multiple regulatory pathways in vitro and ex vivo.硼替佐米在多发性骨髓瘤细胞中对端粒酶活性的差异下调——体外和体内的多种调节途径。
Br J Cancer. 2012 Nov 20;107(11):1844-52. doi: 10.1038/bjc.2012.460.
7
KLF9 is a novel transcriptional regulator of bortezomib- and LBH589-induced apoptosis in multiple myeloma cells.KLF9 是一种新型转录调节因子,可调节硼替佐米和 LBH589 诱导的多发性骨髓瘤细胞凋亡。
Blood. 2012 Feb 9;119(6):1450-8. doi: 10.1182/blood-2011-04-346676. Epub 2011 Dec 5.
8
Proteasome regulator marizomib (NPI-0052) exhibits prolonged inhibition, attenuated efflux, and greater cytotoxicity than its reversible analogs.蛋白酶体调节剂马利佐米(NPI-0052)的抑制作用持久,外排减少,细胞毒性比其可逆类似物更强。
J Pharmacol Exp Ther. 2011 May;337(2):479-86. doi: 10.1124/jpet.110.177824. Epub 2011 Feb 8.
9
Proteasome inhibitors activate autophagy as a cytoprotective response in human prostate cancer cells.蛋白酶体抑制剂通过诱导自噬作为一种细胞保护应答激活人前列腺癌细胞。
Oncogene. 2010 Jan 21;29(3):451-62. doi: 10.1038/onc.2009.343. Epub 2009 Nov 2.
10
Proteasome inhibitors induce a terminal unfolded protein response in multiple myeloma cells.蛋白酶体抑制剂在多发性骨髓瘤细胞中诱导终末未折叠蛋白反应。
Blood. 2006 Jun 15;107(12):4907-16. doi: 10.1182/blood-2005-08-3531. Epub 2006 Feb 28.