Dunham C Michael, Sipe Eilynn K, Peluso LeeAnn
Department of Surgery, St, Elizabeth Health Center, Belmont Avenue, Youngstown, OH, USA.
BMC Surg. 2004 Jan 19;4:3. doi: 10.1186/1471-2482-4-3.
We sought to determine torso injury rates and sensitivities associated with fluid-positive abdominal ultrasound, metabolic acidosis (increased base deficit and lactate), and impaired pulmonary physiology (decreased spirometric volume and PaO2/FiO2).
Level I trauma center prospective pilot and post-pilot study (2000-2001) of stable patients. Increased base deficit was < 0.0 in ethanol-negative and < or = -3.0 in ethanol-positive patients. Increased lactate was > 2.5 mmol/L in ethanol-negative and > or = 3.0 mmol/L in ethanol-positive patients. Decreased PaO2/FiO2 was < 350 and decreased spirometric volume was < 1.8 L.
Of 215 patients, 66 (30.7%) had a torso injury (abdominal/pelvic injury n = 35 and/or thoracic injury n = 43). Glasgow Coma Scale score was 14.8 +/- 0.5 (13-15). Torso injury rates and sensitivities were: abdominal ultrasound negative and normal base deficit, lactate, PaO2/FiO2, and spirometric volume--0.0% & 0.0%; normal base deficit and normal spirometric volume--4.2% & 4.5%; chest/abdominal soft tissue injury--37.8% & 47.0%; increased lactate--39.7% & 47.0%; increased base deficit--41.3% & 75.8%; increased base deficit and/or decreased spirometric volume--43.8% & 95.5%; decreased PaO2/FiO2--48.9% & 33.3%; positive abdominal ultrasound--62.5% & 7.6%; decreased spirometric volume--73.4% & 71.2%; increased base deficit and decreased spirometric volume--82.9% & 51.5%.
Trauma patients with normal base deficit and spirometric volume are unlikely to have a torso injury. Patients with increased base deficit or lactate, decreased spirometric volume, decreased PaO2/FiO2, or positive FAST have substantial risk for torso injury. Increased base deficit and/or decreased spirometric volume are highly sensitive for torso injury. Base deficit and spirometric volume values are readily available and increase or decrease the suspicion for torso injury.
我们试图确定与腹部超声显示有积液、代谢性酸中毒(碱缺失增加和乳酸升高)以及肺生理功能受损(肺活量和动脉血氧分压/吸入氧分数值降低)相关的躯干损伤发生率及敏感性。
对一级创伤中心2000 - 2001年收治的病情稳定患者进行前瞻性试点研究及试点后研究。乙醇阴性患者碱缺失增加定义为<0.0,乙醇阳性患者为≤ - 3.0;乙醇阴性患者乳酸升高定义为>2.5 mmol/L,乙醇阳性患者为≥3.0 mmol/L;动脉血氧分压/吸入氧分数值降低定义为<350,肺活量降低定义为<1.8 L。
215例患者中,66例(30.7%)有躯干损伤(腹部/盆腔损伤35例和/或胸部损伤43例)。格拉斯哥昏迷量表评分为14.8±0.5(13 - 15分)。躯干损伤发生率及敏感性如下:腹部超声阴性且碱缺失、乳酸、动脉血氧分压/吸入氧分数值及肺活量正常——0.0%和0.0%;碱缺失正常且肺活量正常——4.2%和4.5%;胸/腹部软组织损伤——37.8%和47.0%;乳酸升高——39.7%和47.0%;碱缺失增加——41.3%和75.8%;碱缺失增加和/或肺活量降低——43.8%和95.5%;动脉血氧分压/吸入氧分数值降低——48.9%和33.3%;腹部超声阳性——62.5%和7.6%;肺活量降低——73.4%和71.2%;碱缺失增加且肺活量降低——82.9%和51.5%。
碱缺失和肺活量正常的创伤患者躯干损伤可能性不大。碱缺失或乳酸升高、肺活量降低、动脉血氧分压/吸入氧分数值降低或腹部超声检查阳性的患者有较高的躯干损伤风险。碱缺失增加和/或肺活量降低对躯干损伤高度敏感。碱缺失和肺活量值易于获取,可增加或降低对躯干损伤的怀疑程度。