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Chemical alterations induced in mouse liver following a single feeding of carbon tetrachloride.

作者信息

TSUBOI K K, STOWELL R E, LEE C S

出版信息

Cancer Res. 1951 Feb;11(2):87-93.

PMID:14812432
Abstract
摘要

相似文献

1
Chemical alterations induced in mouse liver following a single feeding of carbon tetrachloride.单次喂食四氯化碳后小鼠肝脏中诱导的化学变化。
Cancer Res. 1951 Feb;11(2):87-93.
2
Enzyme alterations associated with mouse liver degeneration and regeneration after single carbon tetrachloride feeding.单次给予四氯化碳后,与小鼠肝脏变性和再生相关的酶改变。
Cancer Res. 1951 Mar;11(3):221-8.
3
Time course of biochemical and histological alterations following a single feeding of carbon tetrachloride to mice.给小鼠单次喂食四氯化碳后生化和组织学改变的时间进程。
Jpn J Exp Med. 1983 Aug;53(4):199-209.
4
Histochemical studies of mouse liver after single feeding of carbon tetrachloride.单次喂食四氯化碳后小鼠肝脏的组织化学研究。
AMA Arch Pathol. 1950 Nov;50(5):519-37.
5
Hepatoprotective effect of Epaltes divaricata extract on carbon tetrachloride induced hepatotoxicity in mice.蟛蜞菊提取物对四氯化碳诱导的小鼠肝毒性的保肝作用。
Indian J Med Res. 2004 Jul;120(1):30-4.
6
[Effects of cholesterol feeding on cholesterol metabolism in rabbits intoxicated with carbon tetrachloride: variations of serum protein and cholesterol patterns in various partitions of serum protein after cholesterol feeding of rabbits intoxicated with carbon tetrachloride].[胆固醇喂养对四氯化碳中毒家兔胆固醇代谢的影响:四氯化碳中毒家兔胆固醇喂养后血清蛋白各组分中血清蛋白和胆固醇模式的变化]
Tip Fak Mecm. 1954;17(1):24-37.
7
TOLERANCE TO CARBON TETRACHLORIDE IN CARBON TETRACHLORIDE-INDUCED CIRRHOSIS.四氯化碳诱导的肝硬化对四氯化碳的耐受性
J Pathol Bacteriol. 1965 Apr;89:611-7.
8
[EXAMINATION OF SOME ENZYME ACTIVITIES IN THE MOUSE LIVER AND KIDNEY BEFORE AND AFTER CARBON TETRACHLORIDE OR URANYL NITRATE ADMINISTRATION].[四氯化碳或硝酸铀酰给药前后小鼠肝脏和肾脏中某些酶活性的检测]
C R Seances Soc Biol Fil. 1963 Dec 15;157:1439-41.
9
A biochemical and immunohistochemical study of the protective effects of carnosine for carbon tetrachloride induced liver injury in rats.肌肽对四氯化碳诱导的大鼠肝损伤保护作用的生化与免疫组化研究
Biotech Histochem. 2015;90(8):608-14. doi: 10.3109/10520295.2015.1044565. Epub 2015 May 15.
10
Metallothionein expression protects against carbon tetrachloride-induced hepatotoxicity, but overexpression and dietary zinc supplementation provide no further protection in metallothionein transgenic and knockout mice.金属硫蛋白的表达可保护机体免受四氯化碳诱导的肝毒性,但在金属硫蛋白转基因和基因敲除小鼠中,其过表达和膳食锌补充并未提供进一步的保护。
J Nutr. 2001 Feb;131(2):215-22. doi: 10.1093/jn/131.2.215.

引用本文的文献

1
On virus metabolism and neoplasia.论病毒代谢与肿瘤形成
Br Med J. 1960 Jul 23;2(5194):252-6. doi: 10.1136/bmj.2.5194.252.
2
HEPATIC PARENCHYMAL CELL ALTERATIONS AFTER LONG-TERM CARBON TETRACHLORIDE ADMINISTRATION. A LIGHT AND ELECTRON MICROSCOPIC STUDY.长期给予四氯化碳后肝实质细胞的改变。光镜与电镜研究
Am J Pathol. 1963 Nov;43(5):867-95.
3
[Serum enzymes as indicators of experimental carbon tetrachloride damage and its therapeutic modification].[血清酶作为实验性四氯化碳损伤及其治疗性改变的指标]
Klin Wochenschr. 1962 Oct 1;40:995-1002. doi: 10.1007/BF01481341.
4
A cytochemical study of the adrenal cortex regenerating after enucleation.摘除术后肾上腺皮质再生的细胞化学研究。
J Physiol. 1957 Mar 11;135(3):536-49. doi: 10.1113/jphysiol.1957.sp005728.
5
Changes in the activities of urea-cycle enzymes after the administration of carbon tetrachloride.四氯化碳给药后尿素循环酶活性的变化。
Biochem J. 1964 May;91(2):261-70. doi: 10.1042/bj0910261.