Bovim Gunnar
Department of Neurology, Trondheim University Hospitals, Regionsykehuset, 7006 TrondheimNorway.
Pain. 1992 Nov;51(2):169-173. doi: 10.1016/0304-3959(92)90258-D.
Pressure-pain threshold (PPT) measurements were performed with a pressure algometer, at 22 specified points in the head in patients with cervicogenic headache (n = 32), migraine (with and without aura) (n = 26) and tension-type headache (n = 17). Comparisons were made with a group of healthy controls (n = 20). The average PPT differed significantly between the groups (ANOVA, F = 9.5, P < 0.0005), largely caused by the low threshold in cervicogenic headache patients. There were no significant differences between controls and the 2 other headache groups. In the cervicogenic headache group, the lowest PPT was found in the occipital part of the head on the side with pain predominance. The ratio between the dominant and non-dominant sides (all 11 points on each side) was 0.85 in cervicogenic headache, whereas it was 0.99 in migraine patients with side preponderance of the pain. The present results support the view that the pathogenesis of cervicogenic headache differs from that of migraine and tension-type headache. The results may further support the theory that fibres from the C2 level (innervating the occipital part of the head) may be included in the pathogenetic mechanism in cervicogenic headache.
使用压力痛觉计对32例颈源性头痛患者、26例偏头痛（有先兆和无先兆）患者以及17例紧张型头痛患者头部的22个特定点进行压痛阈（PPT）测量，并与20名健康对照者进行比较。各组间平均PPT差异显著（方差分析，F = 9.5，P < 0.0005），主要是由于颈源性头痛患者的阈值较低。对照组与其他两组头痛患者之间无显著差异。在颈源性头痛组中，疼痛优势侧头部枕部的PPT最低。颈源性头痛患者优势侧与非优势侧（每侧各11个点）的比值为0.85，而疼痛侧优势的偏头痛患者该比值为0.99。目前的结果支持颈源性头痛的发病机制不同于偏头痛和紧张型头痛的观点。这些结果可能进一步支持这样一种理论，即来自C2水平（支配头部枕部）的纤维可能参与了颈源性头痛的发病机制。
