利用酵母富含内质网的膜组分重建从内质网到高尔基体复合体的转运过程。 - Suppr

Reconstitution of transport from endoplasmic reticulum to Golgi complex using endoplasmic reticulum-enriched membrane fraction from yeast.

作者信息

Wuestehube L J, Schekman R W

机构信息

Division of Biochemistry and Molecular Biology, Howard Hughes Medical Institute, University of California, Berkeley 94720.

出版信息

Methods Enzymol. 1992;219:124-36. doi: 10.1016/0076-6879(92)19015-x.

DOI:10.1016/0076-6879(92)19015-x

PMID:1487986

Abstract

摘要

相似文献

Reconstitution of transport from endoplasmic reticulum to Golgi complex using endoplasmic reticulum-enriched membrane fraction from yeast.利用酵母富含内质网的膜组分重建从内质网到高尔基体复合体的转运过程。

Methods Enzymol. 1992;219:124-36. doi: 10.1016/0076-6879(92)19015-x.

Reconstitution of endoplasmic reticulum to Golgi transport in yeast: in vitro assay to characterize secretory mutants and functional transport vesicles.酵母中内质网到高尔基体转运的重建：用于鉴定分泌突变体和功能性转运囊泡的体外分析

Methods Enzymol. 1992;219:137-52. doi: 10.1016/0076-6879(92)19016-y.

Purification of yeast Sec23 protein by complementation of mutant cell lysates deficient in endoplasmic reticulum-to-Golgi transport.

Methods Enzymol. 1992;219:338-52. doi: 10.1016/0076-6879(92)19034-4.

Isolation of a functional vesicular intermediate that mediates ER to Golgi transport in yeast.在酵母中分离出一种介导内质网到高尔基体运输的功能性囊泡中间体。

J Cell Biol. 1990 Jul;111(1):45-53. doi: 10.1083/jcb.111.1.45.

Use of sec mutants to define intermediates in protein transport from endoplasmic reticulum.利用分泌缺陷型突变体来确定蛋白质从内质网转运过程中的中间体。

Methods Enzymol. 1992;219:267-86. doi: 10.1016/0076-6879(92)19028-5.

Reconstitution of SEC gene product-dependent intercompartmental protein transport.SEC基因产物依赖性的隔室间蛋白质转运的重建。

Cell. 1988 Jul 29;54(3):335-44. doi: 10.1016/0092-8674(88)90196-1.

Reconstitution of protein transport from the endoplasmic reticulum to the Golgi using a cell free system.利用无细胞系统重建从内质网到高尔基体的蛋白质运输。

Prog Clin Biol Res. 1988;270:333-42.

Reconstitution of protein transport from the endoplasmic reticulum to the Golgi complex in yeast: the acceptor Golgi compartment is defective in the sec23 mutant.酵母中蛋白质从内质网到高尔基体复合体转运的重建：在sec23突变体中，受体高尔基体区室存在缺陷。

J Cell Biol. 1988 Oct;107(4):1465-76. doi: 10.1083/jcb.107.4.1465.

Yeast vacuolar proenzymes are sorted in the late Golgi complex and transported to the vacuole via a prevacuolar endosome-like compartment.酵母液泡前体酶在高尔基体晚期复合体中进行分选，并通过类似前液泡内体的区室转运至液泡。

J Cell Biol. 1993 Jun;121(6):1245-56. doi: 10.1083/jcb.121.6.1245.

Use of two-stage incubations to define sequential intermediates in endoplasmic reticulum to Golgi transport.

Methods Enzymol. 1992;219:261-7. doi: 10.1016/0076-6879(92)19027-4.

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Yeast Svf1 binds ceramides and contributes to sphingolipid metabolism at the ER cis-Golgi interface.酵母 Svf1 结合神经酰胺，并有助于内质网-高尔基体 cis-顺面界区的神经鞘脂代谢。

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Structure of the complete, membrane-assembled COPII coat reveals a complex interaction network.完整膜组装的 COPII 衣壳结构揭示了复杂的相互作用网络。

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Vps39 is required for ethanolamine-stimulated elevation in mitochondrial phosphatidylethanolamine.Vps39 对于乙醇胺刺激的线粒体磷酯酰乙醇胺升高是必需的。

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Molecular dissection of the Erv41-Erv46 retrograde receptor reveals a conserved cysteine-rich region in Erv46 required for retrieval activity.Erv41-Erv46逆向受体的分子剖析揭示了Erv46中一个对回收活性至关重要的保守富半胱氨酸区域。

Mol Biol Cell. 2020 Feb 1;31(3):209-220. doi: 10.1091/mbc.E19-08-0484. Epub 2019 Dec 11.

Oxysterol-binding protein homologs mediate sterol transport from the endoplasmic reticulum to mitochondria in yeast.氧化固醇结合蛋白同源物介导固醇从内质网到酵母线粒体的运输。

J Biol Chem. 2018 Apr 13;293(15):5636-5648. doi: 10.1074/jbc.RA117.000596. Epub 2018 Feb 27.

The Golgin protein Coy1 functions in intra-Golgi retrograde transport and interacts with the COG complex and Golgi SNAREs.高尔基体蛋白Coy1在高尔基体内部逆行运输中发挥作用，并与COG复合物和高尔基体SNARE蛋白相互作用。

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Two novel effectors of trafficking and maturation of the yeast plasma membrane H -ATPase.两种新型效应物参与酵母质膜 H+-ATPase 的运输和成熟。

Traffic. 2017 Oct;18(10):672-682. doi: 10.1111/tra.12503. Epub 2017 Aug 16.

The SND proteins constitute an alternative targeting route to the endoplasmic reticulum.SND蛋白构成了一条通向内质网的替代靶向途径。

Nature. 2016 Nov 30;540(7631):134-138. doi: 10.1038/nature20169.

Overexpression of Sly41 suppresses COPII vesicle-tethering deficiencies by elevating intracellular calcium levels.Sly41的过表达通过提高细胞内钙水平来抑制COPII囊泡拴系缺陷。

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Reconstitution of transport from endoplasmic reticulum to Golgi complex using endoplasmic reticulum-enriched membrane fraction from yeast.

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