Akimoto T, Yagi Y, Shikawa A, Yamazaki K, Endo M, Koyanagi H
Department of Cardiovascular Surgery, Heart Institute of Japan, Tokyo.
Nihon Kyobu Geka Gakkai Zasshi. 1992 Dec;40(12):2241-6.
A 38-year-old man with total repair of tetralogy of Fallot at the age of 16 suffered from paroxysmal ventricular tachycardia. His first attack of sustained ventricular tachycardia was recognized at the age of 37 and it was refractory for medical therapy. Electrophysiologic study demonstrated two morphological types of clinical ventricular tachycardias, one originated from the outflow tract of the right ventricle and the other from the area around the patch for closure of ventricular septal defect. He underwent cryosurgical ablation for ventricular tachycardia and patch-closure for residual shunt of ventricular septal defect following the failure of electrical ablation. All of clinical ventricular tachycardias disappeared postoperatively without antiarrhythmic drugs.
一名38岁男性,16岁时接受了法洛四联症的完全修复手术,现患有阵发性室性心动过速。他在37岁时首次出现持续性室性心动过速,药物治疗无效。电生理研究显示临床室性心动过速有两种形态类型,一种起源于右心室流出道,另一种起源于室间隔缺损修补片周围区域。在电消融失败后,他接受了室性心动过速的冷冻手术消融以及室间隔缺损残余分流的补片闭合手术。术后所有临床室性心动过速均消失,无需使用抗心律失常药物。