Baur X
Ordinariat und Zentralinstitut für Arbeitsmedizin, Hamburg.
Pneumologie. 2004 Feb;58(2):107-15. doi: 10.1055/s-2003-812514.
The long-term exposure to dust in the hard coal mining industry can lead to various pathological lung changes, especially to chronic bronchitis without and with obstructive ventilation disorder, lung emphysema, pneumoconiosis (coal miner's pneumoconiosis, in Germany categorized as silicosis) and silicotuberculosis. These health disorders show a close pathogenetic and pathophysiological association and should not necessarily be regarded as individual entities. Most exposed subjects demonstrate more or less all of these pathological disorders. On account of individual (genetic?) susceptibility, their degree differs greatly. Some individuals are largely resistent, other subjects show severe effects like emphysema, progressive massive pneumoconiosis, or the Caplan syndrome. Several studies showed that the pathologically verified degree of lung fibrosis is associated with lung crystalline SiO(2) content whereas the emphysema score is inversely correlated with the coal content. With regard to diagnostics and medical expert opinion, it is important that conventional radiology has a low sensitivity. Further, health impairments of miners engaged for longtime which are insurance relevant (MdE) exist in cases without (BK 4111 if beginning after 12/31/1992) or with coalworkers' pneumoconiosis even for categories < 2/3.
长期接触硬煤采矿业中的粉尘会导致各种肺部病理变化,尤其是导致无阻塞性通气障碍和有阻塞性通气障碍的慢性支气管炎、肺气肿、尘肺病(煤矿工人尘肺病,在德国归类为矽肺病)以及矽肺结核。这些健康问题呈现出密切的发病机制和病理生理关联,不一定应被视为独立的个体疾病。大多数接触者或多或少都有所有这些病理疾病。由于个体(遗传?)易感性,其程度差异很大。一些个体具有很大的抵抗力,而其他个体则表现出严重的影响,如肺气肿、进行性大块纤维化或卡普兰综合征。多项研究表明,经病理证实的肺纤维化程度与肺中结晶二氧化硅含量相关,而肺气肿评分与煤含量呈负相关。关于诊断和医学专家意见,传统放射学敏感性较低这一点很重要。此外,即使对于<2/3类别的情况,长期从事采矿工作且与保险相关(职业病)的矿工,在没有(如果1992年12月31日之后开始则为BK 4111)或患有煤工尘肺病的情况下也存在健康损害。