Vaphiades Michael S
Department of Ophthalmology, University of Alabama, 700 South 18th Street, Birmingham, AL 53233, USA.
J Neuroophthalmol. 2004 Sep;24(3):235-6. doi: 10.1097/00041327-200409000-00011.
A 57-year-old woman who had hypotension and cardiac arrest during coronary artery bypass grafting developed hypotensive ischemic optic neuropathy with no light perception vision OU. Bilateral mid-orbital optic nerve enhancement was found on magnetic resonance imaging (MRI) eight weeks after surgery. Re-examination 16 weeks after surgery showed no light perception vision, dilated un-reactive pupils, and pale optic discs. Bilateral optic nerve enhancement persisted on MRI. Optic nerve enhancement has been reported commonly in radiation-induced ischemic optic neuropathy, occasionally in arteritic ischemic optic neuropathy, and rarely in nonarteritic ischemic optic neuropathy. It has never been reported in hypotensive ischemic optic neuropathy.
一名57岁女性在冠状动脉搭桥手术期间出现低血压和心脏骤停,术后发展为双眼无光感的低血压性缺血性视神经病变。术后8周的磁共振成像(MRI)显示双侧眶中部视神经强化。术后16周复查显示双眼无光感、瞳孔散大固定及视盘苍白。MRI显示双侧视神经强化持续存在。视神经强化常见于放射性缺血性视神经病变,偶见于动脉炎性缺血性视神经病变,在非动脉炎性缺血性视神经病变中罕见。低血压性缺血性视神经病变中从未有过相关报道。