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伯氏疏螺旋体B31的BBE02缺失突变体具有高度可转化的感染性表型。

BBE02 disruption mutants of Borrelia burgdorferi B31 have a highly transformable, infectious phenotype.

作者信息

Kawabata Hiroki, Norris Steven J, Watanabe Haruo

机构信息

Department of Bacteriology, National Institute of Infectious Diseases, Toyama 1-23-1, Shinjuku-ku, Tokyo 162-8640, Japan.

出版信息

Infect Immun. 2004 Dec;72(12):7147-54. doi: 10.1128/IAI.72.12.7147-7154.2004.

Abstract

We constructed highly transformable and infectious Borrelia burgdorferi B31 by inactivating BBE02, a putative restriction-modification gene on the linear plasmid lp25. The low-passage-number B31 clones 5A4 (containing all plasmids) and 5A18 (lp28-4(-) lp56(-)) were used for this study, and BBE02 was disrupted by homologous recombination. The transformation efficiency with the shuttle vector pBSV2C03::gntDeltakan was increased from <1 to approximately 10 colonies per mug of DNA for 5A4 and 5A4 BBE02::Kan(r) and from 14 to approximately 600 colonies per mug of DNA for 5A18 and 5A18 BBE02::Kan(r). lp25, which is required for infectivity in mice, was retained in BBE02 mutants transformed with pBSV2C03::gntDeltakan, but lp25 was not detected in transformants of the parental clones 5A4 and 5A18. BBE02 disruptants and pBSV2C03::gntDeltakan transformants of these clones remained infectious in C3H/HeN mice, and the 50% infective doses of the BBE02 disruptants were <10(2) organisms per mouse. The inactivation of BBE02 thus eliminates a transformation barrier for infectious B. burgdorferi B31 and will provide a valuable tool for studying the virulence factors of Lyme disease.

摘要

我们通过使线性质粒lp25上的一个假定的限制修饰基因BBE02失活,构建了具有高转化能力和感染性的伯氏疏螺旋体B31。本研究使用了低传代次数的B31克隆5A4(包含所有质粒)和5A18(lp28 - 4(-) lp56(-)),通过同源重组破坏BBE02。对于5A4和5A4 BBE02::Kan(r),使用穿梭载体pBSV2C03::gntDeltakan的转化效率从每微克DNA <1个菌落提高到约10个菌落;对于5A18和5A18 BBE02::Kan(r),从每微克DNA 14个菌落提高到约600个菌落。在用pBSV2C03::gntDeltakan转化的BBE02突变体中保留了小鼠感染性所需的lp25,但在亲本克隆5A4和5A18的转化子中未检测到lp25。这些克隆的BBE02破坏体和pBSV2C03::gntDeltakan转化子在C3H/HeN小鼠中仍具感染性,BBE破坏体的50%感染剂量为每只小鼠<10(2)个生物体。因此,BBE02的失活消除了感染性伯氏疏螺旋体B31的转化障碍,并将为研究莱姆病的毒力因子提供有价值的工具。

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