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患有神经性(夏科氏)关节病患者在全接触石膏固定期间的骨密度

Bone mineral density during total contact cast immobilization for a patient with neuropathic (Charcot) arthropathy.

作者信息

Hastings Mary K, Sinacore David R, Fielder Faye A, Johnson Jeffrey E

机构信息

Program in Physical Therapy, School of Medicine, Washington University, St Louis, MO 63110, USA.

出版信息

Phys Ther. 2005 Mar;85(3):249-56.

Abstract

BACKGROUND AND PURPOSE

Diabetes mellitus (DM)-related neuropathic arthropathy of the foot is a destructive bone and joint process. The effect of cast immobilization and non-weight bearing on bone loss has not been well studied. The purpose of this case report is to describe the changes in bone mineral density (BMD) of the calcaneus in the feet of a patient with acute neuropathic arthropathy during total contact cast immobilization.

CASE DESCRIPTION

The patient was a 34-year-old woman with type 1 DM, renal failure requiring dialysis, and a 7-week duration of neuropathic arthropathy of the midfoot. Intervention included total contact casting and minimal to no weight bearing for 10 weeks, with transition to therapeutic footwear. Ultrasound-derived estimates of BMD were taken of both involved and uninvolved calcanei.

OUTCOME

Bone mineral density decreased for the involved foot (from 0.25 g/cm(2) to 0.20 g/cm(2)) and increased for the uninvolved foot (from 0.27 g/cm(2) to 0.31 g/cm(2)) during casting.

DISCUSSION

The low initial BMD and further loss during casting suggest the need for transitional bracing and a well-monitored return to full activity to minimize the risk of recurrence and progression of foot deformity.

摘要

背景与目的

糖尿病(DM)相关的足部神经性关节病是一种破坏性的骨与关节病变。石膏固定和不负重对骨质流失的影响尚未得到充分研究。本病例报告的目的是描述一名急性神经性关节病患者在全接触石膏固定期间足部跟骨骨密度(BMD)的变化。

病例描述

患者为一名34岁的1型糖尿病女性,因肾衰竭需要透析,患有中足神经性关节病7周。干预措施包括全接触石膏固定和10周内尽量少负重或不负重,之后过渡到治疗性鞋具。对患侧和未患侧跟骨进行了超声测定骨密度。

结果

在石膏固定期间,患侧足部骨密度降低(从0.25克/平方厘米降至0.20克/平方厘米),未患侧足部骨密度升高(从0.27克/平方厘米升至0.31克/平方厘米)。

讨论

初始骨密度较低以及石膏固定期间进一步流失表明,需要进行过渡性支具固定,并在密切监测下逐步恢复完全活动,以尽量降低足部畸形复发和进展的风险。

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