Ejima Koichiro, Uchida Tatsuro, Hen Yasuki, Nishio Yukiko, Nomoto Fumiko, Uchida Yoshie, Suzuki Akira, Sato Takahiro, Tanino Shunsuke
Department of Cardiology, Sendai Cardiovascular Center, Sendai.
J Cardiol. 2005 Jul;46(1):33-7.
A 62-year-old woman was admitted to our hospital because of fever in August 2002. She had been treated under a diagnosis of Eisenmenger syndrome with ventricular septal defect since 1988. On admission, echocardiography and color Doppler echocardiography revealed a markedly enlarged pulmonary artery with a mobile flap, and dissection of the pulmonary artery. The origin of the fever could not be identified, and the fever subsided spontaneously without specific treatment. She had no chest pain, but fever might have been a sign of dissection in this patient. Longstanding pulmonary hypertension may cause dissection, which may lead to sudden death or pulmonary hemorrhage often seen in patients with Eisenmenger syndrome. Our patient was a rare survivor without serious bleeding complication.
一名62岁女性于2002年8月因发热入住我院。自1988年起,她一直被诊断为患有室间隔缺损的艾森曼格综合征并接受治疗。入院时,超声心动图和彩色多普勒超声心动图显示肺动脉明显增粗,伴有可移动的瓣膜,以及肺动脉夹层。发热原因不明,未经特殊治疗发热自行消退。她没有胸痛,但发热可能是该患者夹层的一个迹象。长期肺动脉高压可能导致夹层,进而可能导致猝死或艾森曼格综合征患者常见的肺出血。我们的患者是罕见的幸存者,没有严重出血并发症。