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[室上性心动过速作为冠状动脉正常患者肌钙蛋白I升高的原因]

[Supraventricular tachycardia as a cause of elevated troponin I in a patient with normal coronary arteries].

作者信息

Elikowski Waldemar, Baszko Artur, Wróblewski Dariusz, Słomczyński Marek, Małek Małgorzata

机构信息

Oddział Chorób Wewnetrznych Szpitala Miejskiego ZOZ Poznań-Nowe Miasto.

出版信息

Pol Merkur Lekarski. 2005 Jun;18(108):696-9.

Abstract

The authors describe otherwise healthy 69-year-old man admitted to the hospital with supraventricular tachycardia (193/min) accompanied by chest pain and marked ST depression on ECG. Sinus rhythm was restored after verapamil (5 mg i.v.) administration. Symptoms resolved and ECG returned to normal. However, elevation of troponin I level (1.2 ng/ml) was observed a few hours later. Echocardiography as well as coronary angiogram were normal. In electrophysiologic study typical AVNRT was diagnosed and successful ablation of the slow pathway was performed. The authors present a review of current literature on myocardial ischaemia due to supraventricular tachycardia despite normal coronary arteries. In conclusion authors suggest that elevated troponin levels may reflect subendocardial ischaemia and should prompt RF ablation of underlying arrhythmia.

摘要

作者描述了一名69岁原本健康的男性因室上性心动过速(193次/分钟)伴胸痛及心电图显示明显ST段压低而入院。静脉注射维拉帕米(5毫克)后恢复窦性心律。症状缓解,心电图恢复正常。然而,数小时后观察到肌钙蛋白I水平升高(1.2纳克/毫升)。超声心动图及冠状动脉造影均正常。在电生理研究中诊断为典型房室结折返性心动过速,并成功进行了慢径路消融。作者对当前关于尽管冠状动脉正常但室上性心动过速导致心肌缺血的文献进行了综述。总之,作者认为肌钙蛋白水平升高可能反映心内膜下缺血,应促使对潜在心律失常进行射频消融。

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