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丝虫对线虫的免疫调节作用。

Immunomodulation by filarial nematodes.

作者信息

Hoerauf A, Satoguina J, Saeftel M, Specht S

机构信息

Institute for Medical Parasitology, University Clinic Bonn, Sigmund Freud Strasse 25, 53105 Bonn, Germany.

出版信息

Parasite Immunol. 2005 Oct-Nov;27(10-11):417-29. doi: 10.1111/j.1365-3024.2005.00792.x.

Abstract

In order to chronically infect their hosts, filarial nematodes have generated a range of strategies to evade and down-modulate the host's immune system. The recent concept of suppression of immune responses by regulatory T cells has in part benefited from examinations in human and murine filariasis. Its further development in basic immunology animal models has in turn helped to better understand down-regulatory immune mechanisms in filariasis. Thus, filarial nematodes orchestrate down-regulation by inducing regulatory T cells and alternatively activated macrophages, which are able to suppress both Th1 and Th2 responses. Regulatory T cells can also induce the secretion of IgG4 from B cells as another arm of modulation. Dendritic cells are down-regulated upon first encounter with infective L3 larvae. Failure to respond to down-regulatory induction is based on genetic traits in hosts and leads to reduced parasite loads, albeit at the expense of pathology and disease. Since down-regulation in chronically and heavily infected hosts extends to third-party antigens, it is essential to analyse the impact of filarial infection for vaccination, allergy and important coinfections such as malaria, in order to foresee and avert potentially disastrous consequences of filariasis control programmes.

摘要

为了长期感染宿主,丝虫线虫已产生一系列策略来逃避和下调宿主的免疫系统。调节性T细胞抑制免疫反应这一最新概念部分得益于对人类和鼠类丝虫病的研究。其在基础免疫学动物模型中的进一步发展反过来又有助于更好地理解丝虫病中的下调免疫机制。因此,丝虫线虫通过诱导调节性T细胞和替代性活化巨噬细胞来精心安排下调过程,这些细胞能够抑制Th1和Th2反应。调节性T细胞还可诱导B细胞分泌IgG4作为调节的另一途径。树突状细胞在首次接触感染性L3幼虫时会被下调。无法对下调诱导作出反应是基于宿主的遗传特征,并导致寄生虫负荷降低,尽管代价是病理和疾病。由于慢性和重度感染宿主中的下调作用会扩展到第三方抗原,因此分析丝虫感染对疫苗接种、过敏以及疟疾等重要合并感染的影响至关重要,以便预见并避免丝虫病控制计划可能产生的灾难性后果。

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