Suzuki H
University of Ocupational and Environmental Health.
Nihon Rinsho. 1992 Apr;50(4):827-33.
Acute demyelinating polyneuropathy (Guillain-Barré syndrome) among secondary autonomic diseases was described as a representative disorder due to autoimmune mechanism. The possible roles of preceding vital infection, cellular and/or humoral immune abnormalities in developing the disease have been considered. A comparison was made between acute demyelinating polyneuropathy and experimental allergic neuritis in respects of their causes. Polyneuropathy associated with M proteinemia and Rowland's syndrome was also briefly reviewed.
在继发性自主神经疾病中，急性脱髓鞘性多发性神经病（吉兰 - 巴雷综合征）被描述为一种由自身免疫机制引起的典型疾病。先前严重感染、细胞和/或体液免疫异常在该疾病发生过程中可能发挥的作用已得到考虑。对急性脱髓鞘性多发性神经病和实验性过敏性神经炎的病因进行了比较。还简要回顾了与M蛋白血症和罗兰综合征相关的多发性神经病。
