Stimulation of autoimmunity by toll-like receptor ligands.
作者信息
Limmer A, Ganss R, Garbi N, Arnold B, Hämmerling G J
机构信息
German Cancer Research Center (DKFZ), Heidelberg, Tumor Immunology Program, Division of Molecular Immunology, Germany.
出版信息
Ann Rheum Dis. 2005 Nov;64 Suppl 4(Suppl 4):iv15-7. doi: 10.1136/ard.2005.042556.
Abstract
摘要
相似文献
1
Stimulation of autoimmunity by toll-like receptor ligands.Toll样受体配体对自身免疫的刺激作用。
Ann Rheum Dis. 2005 Nov;64 Suppl 4(Suppl 4):iv15-7. doi: 10.1136/ard.2005.042556.
2
Therapeutic targeting of innate immunity with Toll-like receptor agonists and antagonists.使用Toll样受体激动剂和拮抗剂对先天免疫进行治疗性靶向。
Nat Med. 2007 May;13(5):552-9. doi: 10.1038/nm1589.
3
Toll like receptors and autoimmunity: a critical appraisal.Toll样受体与自身免疫：批判性评估
J Autoimmun. 2007 Dec;29(4):310-8. doi: 10.1016/j.jaut.2007.09.001. Epub 2007 Oct 24.
4
[Toll-like receptors recognizing ligands of herpesvirus].识别疱疹病毒配体的Toll样受体
Zh Mikrobiol Epidemiol Immunobiol. 2009 Mar-Apr(2):108-11.
5
Nucleic acid recognizing Toll-like receptors and autoimmunity.识别核酸的Toll样受体与自身免疫
Curr Opin Immunol. 2007 Dec;19(6):606-10. doi: 10.1016/j.coi.2007.10.004. Epub 2007 Dec 3.
6
An unexpected role for RNA-sensing toll-like receptors in a murine model of DNA accrual.RNA 传感 Toll 样受体在 DNA 累积小鼠模型中的意外作用。
Clin Exp Rheumatol. 2015 Jul-Aug;33(4 Suppl 92):S70-3. Epub 2015 Oct 12.
7
Controlling systems of nucleic acid sensing-TLRs restrict homeostatic inflammation.核酸感应 TLRs 的调控系统限制了稳态炎症。
Exp Cell Res. 2012 Aug 1;318(13):1461-6. doi: 10.1016/j.yexcr.2012.03.032. Epub 2012 Apr 6.
8
Immune system's Toll-like receptors have good opportunity for cancer treatment.免疫系统的Toll样受体在癌症治疗方面有很好的前景。
J Natl Cancer Inst. 2006 May 3;98(9):574-5. doi: 10.1093/jnci/djj198.
9
Opposing effects of Toll-like receptor stimulation induce autoimmunity or tolerance.Toll样受体刺激的相反作用可诱导自身免疫或耐受。
Trends Immunol. 2007 Feb;28(2):74-9. doi: 10.1016/j.it.2006.12.006. Epub 2007 Jan 2.
10
Altered toll-like receptor responsiveness underlies a dominant heritable defect in B cell tolerance in autoimmune New Zealand Black mice.改变的 Toll 样受体反应性是自身免疫性新西兰黑鼠 B 细胞耐受中显性遗传缺陷的基础。
Eur J Immunol. 2018 Mar;48(3):492-497. doi: 10.1002/eji.201747287. Epub 2018 Jan 19.
本文引用的文献
1
Toll-like receptor engagement converts T-cell autoreactivity into overt autoimmune disease.Toll样受体的激活可将T细胞自身反应性转变为明显的自身免疫性疾病。
Nat Med. 2005 Feb;11(2):138-45. doi: 10.1038/nm1176. Epub 2005 Jan 16.
2
Mini-review: overcoming tumor-intrinsic resistance to immune effector function.小型综述：克服肿瘤对免疫效应功能的内在抗性
Eur J Immunol. 2004 Oct;34(10):2635-41. doi: 10.1002/eji.200425474.
3
CpG motifs as proinflammatory factors render autochthonous tumors permissive for infiltration and destruction.作为促炎因子的CpG基序使原发性肿瘤易于被浸润和破坏。
J Immunol. 2004 May 15;172(10):5861-9. doi: 10.4049/jimmunol.172.10.5861.
4
CpG-ODN-induced inflammation is sufficient to cause T-cell-mediated autoaggression against hepatocytes.CpG寡脱氧核苷酸诱导的炎症足以引发T细胞介导的针对肝细胞的自身攻击。
Eur J Immunol. 2002 Dec;32(12):3628-37. doi: 10.1002/1521-4141(200212)32:12<3628::AID-IMMU3628>3.0.CO;2-E.
5
Combination of T-cell therapy and trigger of inflammation induces remodeling of the vasculature and tumor eradication.T细胞疗法与炎症触发因素的联合可诱导血管重塑并根除肿瘤。
Cancer Res. 2002 Mar 1;62(5):1462-70.
6
Transformation of the microvascular system during multistage tumorigenesis.多阶段肿瘤发生过程中微血管系统的转变。
Int J Cancer. 2002 Feb 20;97(6):719-25. doi: 10.1002/ijc.10074.
7
Tumor microenvironment can restrict the effectiveness of activated antitumor lymphocytes.肿瘤微环境会限制活化抗肿瘤淋巴细胞的有效性。
Cancer Res. 1998 Oct 15;58(20):4673-81.
8
9
Failure to induce organ-specific autoimmunity by breaking of tolerance: importance of the microenvironment.通过打破耐受性未能诱导器官特异性自身免疫：微环境的重要性。
Eur J Immunol. 1998 Aug;28(8):2395-406. doi: 10.1002/(SICI)1521-4141(199808)28:08<2395::AID-IMMU2395>3.0.CO;2-D.
10
The immunopharmacology of head and neck cancer: an update.头颈癌的免疫药理学：最新进展
Int J Immunopharmacol. 1997 Nov-Dec;19(11-12):629-44. doi: 10.1016/s0192-0561(97)00063-5.
Zotero 插件