Nakayama Yukiteru, Ohno Makoto, Yonemura Shigeto, Uozumi Hiroki, Kobayakawa Naoshi, Fukushima Kazuyuki, Takeuchi Hiroaki, Aoyagi Teruhiko
Department of Cardiovascular Medicine, Japanese Red Cross Medical Center, Hiroo, Tokyo, Japan.
Pacing Clin Electrophysiol. 2006 Jan;29(1):106-8. doi: 10.1111/j.1540-8159.2006.00284.x.
A 42-year-old man was admitted to our hospital with palpitation attacks. Holter ECG showed 2:1 atrioventricular block and bradycardia with the minimum heart rate of 44 beats/min. There was a possible indication of electrophysiological study and cardiac pacemaker implantation. Laboratory data on admission revealed elevated thyrotropin level, with normal thyroxine level. To rule out functional atrioventricular block, we tried 2 weeks of the thyroxine supplementation, and Holter ECG showed improved heart rate without any atrioventricular block or long pause. We experienced that subclinical hypothyroidism caused severe bradycardia and 2:1 atrioventricular block, and that thyroxine supplementation completely improved these conditions.
一名42岁男性因心悸发作入院。动态心电图显示2:1房室传导阻滞及心动过缓，最低心率为44次/分钟。有进行电生理研究及植入心脏起搏器的可能指征。入院时实验室检查数据显示促甲状腺激素水平升高，甲状腺素水平正常。为排除功能性房室传导阻滞，我们尝试补充甲状腺素2周，动态心电图显示心率改善，且无任何房室传导阻滞或长间歇。我们发现亚临床甲状腺功能减退可导致严重心动过缓和2:1房室传导阻滞，补充甲状腺素可完全改善这些情况。
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