芳烃受体与二噁英毒性机制 - Suppr

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超能文献

The Ah receptor and the mechanism of dioxin toxicity.

作者信息

Landers J P, Bunce N J

机构信息

Department of Biochemistry and Molecular Biology, Mayo Clinic, Rochester, MN 55905.

出版信息

Biochem J. 1991 Jun 1;276 ( Pt 2)(Pt 2):273-87. doi: 10.1042/bj2760273.

DOI:10.1042/bj2760273

PMID:1646595

原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1151088/

Abstract

摘要

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9b5c/1151088/8aa67037e928/biochemj00158-0014-a.jpg

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The Ah receptor and the mechanism of dioxin toxicity.芳烃受体与二噁英毒性机制

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The role of receptors in 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) toxicity.受体在2,3,7,8-四氯二苯并对二恶英（TCDD）毒性中的作用。

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2,3,7,8,-Tetrachlorodibenzo-p-dioxin: segregation of toxocity with the Ah locus.2,3,7,8-四氯二苯并对二恶英：毒性与芳烃（Ah）位点的分离

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Formation of 2,3,7,8-tetrachlorodibenzodioxin by thermal decomposition of sodium 2,4,5,-trichlorophenate.通过2,4,5-三氯苯甲酸钠的热分解形成2,3,7,8-四氯二苯并二恶英。

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Tissue distribution, excretion, and metabolism of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) in the Golden Syrian hamster.2,3,7,8-四氯二苯并对二噁英（TCDD）在金黄叙利亚仓鼠体内的组织分布、排泄及代谢

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Toxicol Appl Pharmacol. 1981 Nov;61(2):269-76. doi: 10.1016/0041-008x(81)90417-8.

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