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Y-40138是一种多种细胞因子产生调节剂,可预防D-半乳糖胺和脂多糖诱导的肝炎。

Y-40138, a multiple cytokine production modulator, protects against D-galactosamine and lipopolysaccharide-induced hepatitis.

作者信息

Fukuda Tetsuko, Mogami Akira, Tanaka Hideki, Yoshikawa Tsutomu, Hisadome Masao, Komatsu Hirotsugu

机构信息

Marketing and Planning Department, Sales and Marketing Division, Mitsubishi Pharma Corporation, 2-5-6 Awaji-machi, Chuo-ku, Osaka 541-0047, Japan.

出版信息

Life Sci. 2006 Jul 24;79(9):822-7. doi: 10.1016/j.lfs.2006.03.025. Epub 2006 Mar 28.

Abstract

Acute and fulminant liver failure induced by viral hepatitis, alcohol or other hepatotoxic drugs are associated with tumor necrosis factor (TNF) production. D-Galactosamine (D-GalN) and lipopolysaccharide (LPS)-induced liver injury is an experimental model of fulminant hepatic failure. In this model, TNF-alpha plays a central role in the pathogenesis of D-GalN/LPS-induced liver injury in mice. Y-40138, N-[1-(4-[4-(pyrimidin-2-yl)piperazin-1-yl]methyl phenyl)cyclopropyl] acetamide.HCl inhibits TNF-alpha and augments interleukin (IL)-10 production in LPS-injected mice in plasma. In the present study, we examined the effect of Y-40138 on D-GalN/LPS-induced hepatitis. Y-40138 (10mg/kg, i.v.) significantly suppressed TNF-alpha and monocyte chemoattractant protein-1 (MCP-1) production and augmented IL-10 production in plasma. In addition, Y-40138 significantly inhibited TNF-alpha production induced by direct interaction between human T lymphocytes and macrophages. Y-40138 suppressed plasma alanine transaminase (ALT) elevation and improved survival rate in D-GalN/LPS-injected mice, and it is suggested that the protective effect of Y-40138 on hepatitis may be mediated by inhibition of TNF-alpha and MCP-1, and/or augmentation of IL-10. This compound is expected to be a new candidate for treatment of cytokine and/or chemokine-related liver diseases such as alcoholic hepatitis.

摘要

由病毒性肝炎、酒精或其他肝毒性药物引起的急性和暴发性肝衰竭与肿瘤坏死因子(TNF)的产生有关。D-半乳糖胺(D-GalN)和脂多糖(LPS)诱导的肝损伤是暴发性肝衰竭的实验模型。在该模型中,TNF-α在D-GalN/LPS诱导的小鼠肝损伤发病机制中起核心作用。Y-40138,N-[1-(4-[4-(嘧啶-2-基)哌嗪-1-基]甲基苯基)环丙基]乙酰胺盐酸盐可抑制TNF-α并增加LPS注射小鼠血浆中白细胞介素(IL)-10的产生。在本研究中,我们检测了Y-40138对D-GalN/LPS诱导的肝炎的影响。Y-40138(10mg/kg,静脉注射)可显著抑制血浆中TNF-α和单核细胞趋化蛋白-1(MCP-1)的产生,并增加IL-10的产生。此外,Y-40138可显著抑制人T淋巴细胞与巨噬细胞直接相互作用诱导的TNF-α产生。Y-40138可抑制D-GalN/LPS注射小鼠血浆中丙氨酸转氨酶(ALT)升高并提高存活率,提示Y-40138对肝炎的保护作用可能通过抑制TNF-α和MCP-1以及/或增加IL-10来介导。该化合物有望成为治疗细胞因子和/或趋化因子相关肝病(如酒精性肝炎)的新候选药物。

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