丙烯酰胺神经病中肌肉传入神经早期放电的保留
Preservation of early discharge of muscle afferents in acrylamide neuropathy.
作者信息
Sumner A J
出版信息
J Physiol. 1975 Mar;246(2):277-88. doi: 10.1113/jphysiol.1975.sp010890.
Abstract
- Responses to stretch and the 'early discharge' have been studied in acrylamide neuropathy in the expectation that functional block of the nerve terminals would allow these two processes to be dissociated. 2. Of 538 single group I and group II muscle afferents which were nonresponsive to stretch, ninety-nine displayed 'early discharge' which appeared to be identical with that described in normally functioning muscle stretch afferents. 3. Preservation of 'early discharge' in non-responsive fibres indicates these impulses do not arise from excitation of the muscle spindle itself. The evidence indicates that 'early discharge' is usually ephaptically excited by the muscle action potential.
摘要
- 人们对丙烯酰胺神经病中的牵张反应和“早期放电”进行了研究，期望神经末梢的功能阻断能使这两个过程得以分离。2. 在538条对牵张无反应的I组和II组单根肌肉传入纤维中，99条显示出“早期放电”，其似乎与正常功能的肌肉牵张传入纤维中所描述的相同。3. 无反应纤维中“早期放电”的保留表明这些冲动并非源于肌梭本身的兴奋。证据表明，“早期放电”通常由肌肉动作电位经电紧张性兴奋产生。
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