Witmer H
J Virol. 1975 Jun;15(6):1511-3. doi: 10.1128/JVI.15.6.1511-1513.1975.
Messengers for T4 phage-specific deoxynucleoside monophosphate kinase overaccumulated in nonpermissive hosts infected with amber-defective viruses that displayed either the DNA-negative or maturation-defective phenotype. Under both conditions, however, transcription of functional messengers for dihydrofolate reductase followed essentially normal kinetics.
T4噬菌体特异性脱氧核苷单磷酸激酶的信使在感染了表现出DNA阴性或成熟缺陷表型的琥珀突变缺陷病毒的非允许宿主中过度积累。然而,在这两种情况下,二氢叶酸还原酶功能性信使的转录基本上遵循正常动力学。