粘球菌的凝聚缺陷突变体。
Cohesion-defective mutants of Myxococcus xanthus.
作者信息
Bonner Pamela J, Shimkets Lawrence J
机构信息
Department of Microbiology, 527 Biological Sciences Building, University of Georgia, Athens, GA 30602, USA.
出版信息
J Bacteriol. 2006 Jun;188(12):4585-8. doi: 10.1128/JB.00237-06.
Abstract
Cohesion of Myxococcus xanthus cells involves interaction of a cell surface cohesin with a component of the extracellular matrix. In this work, two previously isolated cohesion-defective (fbd) mutants were characterized. The fbdA and fbdB genes do not encode the cohesins but are necessary for their production. Both mutants produce type IV pili, suggesting that PilA is not a major cohesin.
摘要
黄色粘球菌细胞的凝聚涉及细胞表面凝聚素与细胞外基质成分的相互作用。在这项研究中，对两个先前分离出的凝聚缺陷型（fbd）突变体进行了表征。fbdA和fbdB基因并不编码凝聚素，但它们的产生是必需的。两个突变体均产生IV型菌毛，这表明菌毛蛋白A不是主要的凝聚素。
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