McConnell J F, Sparkes A H, Ladlow J, Doust R, Davies S
Animal Health Trust, Newmarket, Lanwades Park, Kentford, Newmarket, Suffolk CB8 7UU, UK.
J Small Anim Pract. 2006 Jun;47(6):338-43. doi: 10.1111/j.1748-5827.2006.00141.x.
Two cases of ascites secondary to portal vascular abnormalities associated with portal hypertension are described. In the first case a five-month-old cat was presented with recurrent ascites and investigations showed that the underlying cause was a hepatic arteriovenous fistula. Ultrasonography showed direct communication of the coeliac artery and right branch of the portal vein. There was also hepatofugal flow in the main portal vein consistent with portal hypertension. The ultrasonographic features were similar to those seen in dogs with hepatic arteriovenous fistulae. In the second case, ascites, portal hypertension and an intraluminal mass in the main portal vein was diagnosed in a 16-year-old cat that had been presented with hyperthyroidism and hepatomegaly. Acquired portosystemic collaterals involving the left renal vein were present. Additional diagnostic investigations were not permitted. Ultrasonography was useful in both cases to document portal hypertension and the underlying cause.
本文描述了两例继发于与门静脉高压相关的门静脉血管异常的腹水病例。第一例是一只五个月大的猫，出现反复腹水，检查显示其根本原因是肝动静脉瘘。超声检查显示腹腔动脉与门静脉右支直接相通。主门静脉中也存在离肝血流，这与门静脉高压一致。超声特征与患有肝动静脉瘘的犬相似。第二例是一只16岁的猫，因甲状腺功能亢进和肝肿大前来就诊，诊断为腹水、门静脉高压和主门静脉内肿块。存在累及左肾静脉的后天性门体分流。未进行进一步的诊断检查。超声检查在这两例病例中对于记录门静脉高压及其根本原因均很有用。
