DL-threo-3-hydroxyaspartate reduces NMDA receptor activation by glutamate in cultured neurons.

Suppr

超能文献

作者信息

Marini A, Novelli A

机构信息

Clinical Neuroscience Branch, National Institute for Neurological Disorders and Stroke (NINDS), Bethesda, MD 20892.

出版信息

Eur J Pharmacol. 1991 Feb 26;194(1):131-2. doi: 10.1016/0014-2999(91)90136-e.

DOI:10.1016/0014-2999(91)90136-e

PMID:1676372

Abstract

摘要

相似文献

DL-threo-3-hydroxyaspartate reduces NMDA receptor activation by glutamate in cultured neurons.

Eur J Pharmacol. 1991 Feb 26;194(1):131-2. doi: 10.1016/0014-2999(91)90136-e.

N-methyl-D-aspartate exposure blocks glutamate toxicity in cultured cerebellar granule cells.N-甲基-D-天冬氨酸处理可阻断培养的小脑颗粒细胞中的谷氨酸毒性。

Mol Pharmacol. 1992 Aug;42(2):210-6.

Excitatory amino acid signal transduction in cerebellar cell cultures.

Funct Neurol. 1986 Oct-Dec;1(4):345-9.

Excitatory amino acid receptors and guanosine 3',5'-cyclic monophosphate in incubated slices of immature and adult rat cerebellum.

Neuroscience. 1982 Oct;7(10):2491-7. doi: 10.1016/0306-4522(82)90209-3.

Basic fibroblast growth factor protects cerebellar neurons in primary culture from NMDA and non-NMDA receptor mediated neurotoxicity.

FEBS Lett. 1993 Nov 29;335(1):124-31. doi: 10.1016/0014-5793(93)80453-2.

Cellular origins of cyclic GMP responses to excitatory amino acid receptor agonists in rat cerebellum in vitro.

J Neurochem. 1987 Jan;48(1):29-39. doi: 10.1111/j.1471-4159.1987.tb13123.x.

N-methyl-D-aspartate receptor agonists decrease protooncogene bcl-2 mRNA expression in cultured rat cerebellar granule neurons.N-甲基-D-天冬氨酸受体激动剂可降低培养的大鼠小脑颗粒神经元中原癌基因bcl-2的mRNA表达。

Biochem Biophys Res Commun. 1993 Sep 15;195(2):623-9. doi: 10.1006/bbrc.1993.2091.

Stimulation of rat cerebellar guanosine 3',5'-cyclic monophosphate (cyclic GMP) levels: effects of amino acid antagonists.大鼠小脑环磷酸鸟苷（cGMP）水平的刺激：氨基酸拮抗剂的作用

Br J Pharmacol. 1981 Nov;74(3):723-9. doi: 10.1111/j.1476-5381.1981.tb10484.x.

Agonist response kinetics of N-methyl-D-aspartate receptors in neurons cultured from rat cerebral cortex and cerebellum: evidence for receptor heterogeneity.大鼠大脑皮层和小脑培养神经元中 N-甲基-D-天冬氨酸受体的激动剂反应动力学：受体异质性的证据

Mol Pharmacol. 1993 Dec;44(6):1252-7.

Glutamate stimulates somatostatin release from diencephalic neurons in primary culture.谷氨酸可刺激原代培养中来自间脑神经元的生长抑素释放。

Endocrinology. 1988 Nov;123(5):2360-6. doi: 10.1210/endo-123-5-2360.

引用本文的文献

A Medicinal Chemistry Perspective on Excitatory Amino Acid Transporter 2 Dysfunction in Neurodegenerative Diseases.兴奋性氨基酸转运体 2 功能障碍与神经退行性疾病的药物化学研究进展

J Med Chem. 2023 Feb 23;66(4):2330-2346. doi: 10.1021/acs.jmedchem.2c01572. Epub 2023 Feb 14.

Preconditioning provides neuroprotection in models of CNS disease: paradigms and clinical significance.预处理为中枢神经系统疾病模型提供神经保护：范例和临床意义。

Prog Neurobiol. 2014 Mar;114:58-83. doi: 10.1016/j.pneurobio.2013.11.005. Epub 2014 Jan 2.

Hormesis, cell death, and regenerative medicine for neurode-generative diseases. hormesis，细胞死亡和神经退行性疾病的再生医学。

Dose Response. 2012 Jul 23;11(2):238-54. doi: 10.2203/dose-response.12-019.Wang. Print 2013.

Cellular and molecular responses of cultured neurons to stressful stimuli.培养神经元对应激刺激的细胞和分子反应。

Dose Response. 2011;9(3):416-33. doi: 10.2203/dose-response.10-041.Marini. Epub 2011 Feb 22.

AMPA prevents glutamate-induced neurotoxicity and apoptosis in cultured cerebellar granule cell neurons.α-氨基-3-羟基-5-甲基-4-异恶唑丙酸（AMPA）可预防培养的小脑颗粒细胞神经元中谷氨酸诱导的神经毒性和细胞凋亡。

Neurotox Res. 2000;2(1):51-61. doi: 10.1007/BF03033327.

N-methyl-D-aspartate receptor-mediated neuroprotection in cerebellar granule cells requires new RNA and protein synthesis.N-甲基-D-天冬氨酸受体介导的小脑颗粒细胞神经保护作用需要新的RNA和蛋白质合成。

Proc Natl Acad Sci U S A. 1992 Jul 15;89(14):6555-9. doi: 10.1073/pnas.89.14.6555.

文献AI研究员

20分钟写一篇综述，助力文献阅读效率提升50倍。

立即体验

用中文搜PubMed

大模型驱动的PubMed中文搜索引擎

马上搜索

文档翻译

学术文献翻译模型，支持多种主流文档格式。

立即体验