在NDEA启动的肝癌发生模型中，乙醇的混合功能氧化酶及促肿瘤作用降低。 - Suppr | 超能文献

A reduction in mixed function oxidases and in tumor promoting effects of ethanol in a NDEA-initiated hepatocarcinogenesis model.

作者信息

Mufti S I, Sipes I G

机构信息

Department of Pharmacology and Toxicology, University of Arizona, College of Pharmacy, Tucson 85721.

出版信息

Adv Exp Med Biol. 1991;283:347-52. doi: 10.1007/978-1-4684-5877-0_45.

DOI:10.1007/978-1-4684-5877-0_45

Abstract

摘要

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A reduction in mixed function oxidases and in tumor promoting effects of ethanol in a NDEA-initiated hepatocarcinogenesis model.在NDEA启动的肝癌发生模型中，乙醇的混合功能氧化酶及促肿瘤作用降低。

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