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Interleukin-27R (WSX-1/T-cell cytokine receptor) gene-deficient mice display enhanced resistance to leishmania donovani infection but develop severe liver immunopathology.
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IL-23 compensates for the absence of IL-12p70 and is essential for the IL-17 response during tuberculosis but is dispensable for protection and antigen-specific IFN-gamma responses if IL-12p70 is available.
J Immunol. 2005 Jul 15;175(2):788-95. doi: 10.4049/jimmunol.175.2.788.
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New IL-12-family members: IL-23 and IL-27, cytokines with divergent functions.
Nat Rev Immunol. 2005 Jul;5(7):521-31. doi: 10.1038/nri1648.
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Interleukin-15 enhances innate and adaptive immune responses to blood-stage malaria infection in mice.
Infect Immun. 2005 May;73(5):3172-7. doi: 10.1128/IAI.73.5.3172-3177.2005.
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Prevention of relapse after chemotherapy in a chronic intracellular infection: mechanisms in experimental visceral leishmaniasis.
J Immunol. 2005 Apr 15;174(8):4916-23. doi: 10.4049/jimmunol.174.8.4916.
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Immunopathogenesis of infection with the visceralizing Leishmania species.
Microb Pathog. 2005 Apr;38(4):147-60. doi: 10.1016/j.micpath.2004.11.002.
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Synergistic activation of interleukin-12 p35 gene transcription by interferon regulatory factor-1 and interferon consensus sequence-binding protein.
J Biol Chem. 2004 Dec 31;279(53):55609-17. doi: 10.1074/jbc.M406565200. Epub 2004 Oct 15.
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The immunopathology of experimental visceral leishmaniasis.
Immunol Rev. 2004 Oct;201:239-53. doi: 10.1111/j.0105-2896.2004.00188.x.
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Immunotherapy with OX40L-Fc or anti-CTLA-4 enhances local tissue responses and killing of Leishmania donovani.
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Interferon-gamma production and host protective response against Mycobacterium tuberculosis in mice lacking both IL-12p40 and IL-18.
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