临床儿茶酚胺神经化学：朱利叶斯·阿克塞尔罗德的遗产。
Clinical catecholamine neurochemistry: a legacy of Julius Axelrod.
作者信息
Goldstein David S, Eisenhofer Graeme, Kopin Irwin J
机构信息
Clinical Neurocardiology Section, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, Maryland 20892-1620, USA.
出版信息
Cell Mol Neurobiol. 2006 Jul-Aug;26(4-6):695-702. doi: 10.1007/s10571-006-9041-0. Epub 2006 Jul 27.
Abstract
- Discoveries, insights, and concepts that Julius Axelrod introduced about the disposition and metabolism of catecholamines provided the scientific basis and spurred the development of clinical catecholamine neurochemistry. 2. Here, we provide examples of this aspect of Axelrod's scientific legacy.
摘要
- 朱利叶斯·阿克塞尔罗德所提出的关于儿茶酚胺处置和代谢的发现、见解及概念，为临床儿茶酚胺神经化学提供了科学依据并推动了其发展。2. 在此，我们给出阿克塞尔罗德科学遗产这一方面的实例。
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本文引用的文献
1
Profound cardiac sympathetic denervation occurs in Parkinson disease.
Brain Pathol. 2005 Jan;15(1):29-34. doi: 10.1111/j.1750-3639.2005.tb00097.x.
2
Diagnosis and localization of pheochromocytoma.
Hypertension. 2004 May;43(5):907-10. doi: 10.1161/01.HYP.0000125014.56023.b8. Epub 2004 Mar 15.
3
Presence of 3-methoxy-4-hydroxy-phenylglycol and metanephrine in phaeochromocytoma tissues.
Nature. 1960 Mar 12;185:788. doi: 10.1038/185788a0.
4
3,4-Dihydroxyphenylglycol, a metabolite of epinephrine.
Arch Biochem Biophys. 1960 Jul;89:148. doi: 10.1016/0003-9861(60)90026-6.
5
TURNOVER AND METABOLISM OF CATECHOLAMINES IN PATIENTS WITH PHEOCHROMOCYTOMA.
J Clin Invest. 1964 Jan;43(1):94-102. doi: 10.1172/JCI104898.
6
Fate of tritiated noradrenaline at the sympathetic nerve-endings.
Nature. 1961 Oct 14;192:172-3. doi: 10.1038/192172a0.
7
Lack of uptake of catecholamines after chronic denervation of sympathetic nerves.
Nature. 1961 Jan 7;189:66. doi: 10.1038/189066a0.
8
Effect of psychotropic drugs on the uptake of H3-norepinephrine by tissues.
Science. 1961 Feb 10;133(3450):383-4. doi: 10.1126/science.133.3450.383.
9
Enzymatic O-methylation of epinephrine and other catechols.
J Biol Chem. 1958 Sep;233(3):702-5.
10
Demonstration of the 3-methoxy analog of norepinephrine in man.
Science. 1958 Apr 18;127(3303):876. doi: 10.1126/science.127.3303.876.
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