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Complement activation during sepsis in humans.

作者信息

Schreiber Heike, Rittirsch Daniel, Flierl Michael, Brueckner Uwe, Schneider Marion, Weiss Manfred, Gebhard Florian, Huber-Lang Markus

机构信息

Dept. of Traumatology, Hand-, Plastic-, and Reconstructive Surgery, Steinhoevelstr.9, 89075 Ulm, Germany.

出版信息

Adv Exp Med Biol. 2006;586:217-26. doi: 10.1007/0-387-34134-X_15.

DOI:10.1007/0-387-34134-X_15
PMID:16893075
Abstract
摘要

相似文献

1
Complement activation during sepsis in humans.人类脓毒症期间的补体激活。
Adv Exp Med Biol. 2006;586:217-26. doi: 10.1007/0-387-34134-X_15.
2
In response to complement anaphylatoxin peptides C3a and C5a, human vascular endothelial cells migrate and mediate the activation of B-cells and polarization of T-cells.为了补充补体过敏毒素肽 C3a 和 C5a,人血管内皮细胞迁移并介导 B 细胞的激活和 T 细胞的极化。
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3
New concepts on the therapeutic control of complement anaphylatoxin receptors.补体过敏毒素受体治疗性控制的新概念。
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4
Food intake regulation by central complement system.中枢补体系统对食物摄入的调节
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5
Plasma clearance of the human C5a anaphylatoxin by binding to leucocyte C5a receptors.通过与白细胞C5a受体结合实现人C5a过敏毒素的血浆清除。
Immunology. 1994 Aug;82(4):516-21.
6
Human plasmacytoid dendritic cells express receptors for anaphylatoxins C3a and C5a and are chemoattracted to C3a and C5a.人类浆细胞样树突状细胞表达过敏毒素C3a和C5a的受体,并被C3a和C5a趋化吸引。
J Invest Dermatol. 2006 Nov;126(11):2422-9. doi: 10.1038/sj.jid.5700416. Epub 2006 Jun 15.
7
Complement Activation in Arterial and Venous Thrombosis is Mediated by Plasmin.补体在动脉和静脉血栓形成中的激活是由纤溶酶介导的。
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Complement peptides and mast cell triggering.补体肽与肥大细胞激活
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9
Complement in sepsis-when science meets clinics.脓毒症中的补体——当科学遇见临床。
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Blockade of C5a and C5b-9 generation inhibits leukocyte and platelet activation during extracorporeal circulation.C5a和C5b-9生成的阻断可抑制体外循环期间白细胞和血小板的激活。
J Clin Invest. 1995 Sep;96(3):1564-72. doi: 10.1172/JCI118195.

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Reduced hemolytic complement activity in the classical pathway (CH50) is a risk factor for poor clinical outcomes of patients with infections: a retrospective analysis of health insurance claims in Japan.经典途径中溶血补体活性降低(CH50)是感染患者临床预后不良的一个危险因素:日本医疗保险理赔的回顾性分析。
Front Immunol. 2025 Jun 5;16:1601690. doi: 10.3389/fimmu.2025.1601690. eCollection 2025.
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Acute respiratory distress syndrome heterogeneity and the septic ARDS subgroup.急性呼吸窘迫综合征异质性和脓毒症相关急性呼吸窘迫综合征亚组。
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Biomarkers for the Prediction and Judgement of Sepsis and Sepsis Complications: A Step towards ?
用于预测和判断脓毒症及脓毒症并发症的生物标志物:迈向?
J Clin Med. 2022 Sep 29;11(19):5782. doi: 10.3390/jcm11195782.
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Increased Alternative Complement Pathway Function and Improved Survival during Critical Illness.补体替代途径功能增强与危重病患者生存改善。
Am J Respir Crit Care Med. 2020 Jul 15;202(2):230-240. doi: 10.1164/rccm.201910-2083OC.
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Biomarkers of sepsis.脓毒症的生物标志物。
Crit Rev Clin Lab Sci. 2013 Jan-Feb;50(1):23-36. doi: 10.3109/10408363.2013.764490.
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A new experimental polytrauma model in rats: molecular characterization of the early inflammatory response.一种新的大鼠多发伤实验模型:早期炎症反应的分子特征。
Mediators Inflamm. 2012;2012:890816. doi: 10.1155/2012/890816. Epub 2012 Feb 29.
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Ann Gen Psychiatry. 2011 May 4;10:16. doi: 10.1186/1744-859X-10-16.
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Complement activation in emergency department patients with severe sepsis.急诊科严重脓毒症患者的补体激活。
Acad Emerg Med. 2010 Apr;17(4):353-9. doi: 10.1111/j.1553-2712.2010.00713.x.