Utilization of in vivo alkylated metabolites as a possible cause of increased sensitivity to tumor cells to alkylating agents.
作者信息
Lerman M I, Abakumova O Y, Kutcenko N G, Sokolova I S, Gorbachieva L B, Serebryannyl A M
出版信息
Mol Biol Rep. 1975 Jul;2(2):151-7. doi: 10.1007/BF00357546.
PMID:169463
Abstract
摘要
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Utilization of in vivo alkylated metabolites as a possible cause of increased sensitivity to tumor cells to alkylating agents.利用体内烷基化代谢物作为肿瘤细胞对烷化剂敏感性增加的可能原因。
Mol Biol Rep. 1975 Jul;2(2):151-7. doi: 10.1007/BF00357546.
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本文引用的文献
1
Sedimentation characteristics of rapidly labelled RNA from HeLa cells.来自海拉细胞的快速标记RNA的沉降特性。
Biochem Biophys Res Commun. 1962 Jun 4;7:486-90. doi: 10.1016/0006-291x(62)90341-8.
2
Proceedings: Tumor angiogenesis factor.会议论文：肿瘤血管生成因子
Cancer Res. 1974 Aug;34(8):2109-13.
3
Different degradation rates of alkylated RNA protein and lipids in normal and tumor cells.正常细胞和肿瘤细胞中烷基化RNA、蛋白质和脂质的不同降解速率。
Cancer Res. 1974 Jul;34(7):1536-41.
4
Patterns of damage and repair of liver DNA induced by carcinogenic methylating agents in vivo.体内致癌甲基化剂诱导的肝脏DNA损伤与修复模式
Cancer Res. 1973 Sep;33(9):2122-8.
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