Dow L E, Kauffman J S, Caddy J, Zarbalis K, Peterson A S, Jane S M, Russell S M, Humbert P O
Cell Cycle & Cancer Genetics Laboratory, Peter MacCallum Cancer Centre, Melbourne, Australia.
Oncogene. 2007 Apr 5;26(16):2272-82. doi: 10.1038/sj.onc.1210016. Epub 2006 Oct 9.
Altered expression of human Scribble is associated with invasive epithelial cancers, however, its role in tumour development remains unclear. Mutations in Drosophila Scribble result in loss of polarity, overproliferation and 3D-tumourous overgrowth of epithelial cells. Using complementation studies in Drosophila we recently demonstrated that expression of human Scribble can also regulate polarity and restrict tissue overgrowth. Here, we have undertaken a detailed study of human Scribble function in the polarized mammary cell line, MCF10A. We show that although Scribble does not seem to be required for apical-basal polarity or proliferation control in MCF10A cells, Scribble is essential for the control of polarity associated with directed epithelial cell migration. Scribble-depleted MCF10A cells show defective in vitro wound closure and chemotactic movement. The cells at the wound edge fail to polarize, show reduced lamellipodia formation and impaired recruitment of Cdc42 and Rac1 to the leading edge. Furthermore, we show that this function is relevant in vivo as Scribble mutant mice show defective epidermal wound healing. This data identifies an essential role for mammalian Scribble in the regulation of the polarity specifically involved in directed epithelial migration.
人类Scribble表达的改变与侵袭性上皮癌相关,然而,其在肿瘤发生中的作用仍不清楚。果蝇Scribble的突变会导致上皮细胞极性丧失、过度增殖和三维肿瘤性过度生长。我们最近利用果蝇的互补研究证明,人类Scribble的表达也可以调节极性并限制组织过度生长。在此,我们对极化乳腺细胞系MCF10A中人类Scribble的功能进行了详细研究。我们发现,虽然Scribble似乎不是MCF10A细胞顶-基极性或增殖控制所必需的,但Scribble对于控制与定向上皮细胞迁移相关的极性至关重要。Scribble缺失的MCF10A细胞在体外伤口愈合和趋化运动方面存在缺陷。伤口边缘的细胞无法极化,片状伪足形成减少,Cdc42和Rac1向前缘的募集受损。此外,我们表明这种功能在体内也相关,因为Scribble突变小鼠表现出表皮伤口愈合缺陷。这些数据确定了哺乳动物Scribble在调节特异性参与定向上皮迁移的极性方面的重要作用。
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