• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

The beat goes on: diastolic noise that just won't quit.

作者信息

Bers Donald M

出版信息

Circ Res. 2006 Oct 27;99(9):921-3. doi: 10.1161/01.RES.0000249859.10103.a9.

DOI:10.1161/01.RES.0000249859.10103.a9
PMID:17068299
Abstract
摘要

相似文献

1
The beat goes on: diastolic noise that just won't quit.搏动仍在继续：舒张期杂音持续不止。
Circ Res. 2006 Oct 27;99(9):921-3. doi: 10.1161/01.RES.0000249859.10103.a9.
2
Membrane potential fluctuations resulting from submembrane Ca2+ releases in rabbit sinoatrial nodal cells impart an exponential phase to the late diastolic depolarization that controls their chronotropic state.兔窦房结细胞中膜下Ca2+释放引起的膜电位波动为控制其变时状态的舒张晚期去极化赋予了一个指数期。
Circ Res. 2006 Oct 27;99(9):979-87. doi: 10.1161/01.RES.0000247933.66532.0b. Epub 2006 Sep 28.
3
A novel mechanism of pacemaker control that depends on high levels of cAMP and PKA-dependent phosphorylation: a precisely controlled biological clock.
Circ Res. 2006 Mar 3;98(4):437-9. doi: 10.1161/01.RES.0000214324.34563.31.
4
Beat-to-beat cycle length variability of spontaneously beating guinea pig sinoatrial cells: relative contributions of the membrane and calcium clocks.豚鼠窦房结细胞自发搏动的逐搏周期长度变异性：膜时钟和钙时钟的相对贡献
PLoS One. 2014 Jun 18;9(6):e100242. doi: 10.1371/journal.pone.0100242. eCollection 2014.
5
Distinguishing properties of cells from the myocardial sleeves of the pulmonary veins: a comparison of normal and abnormal pacemakers.肺静脉心肌袖细胞的鉴别特性：正常与异常起搏点的比较
Circ Arrhythm Electrophysiol. 2008 Apr;1(1):39-48. doi: 10.1161/CIRCEP.107.748467.
6
Diastolic calcium release controls the beating rate of rabbit sinoatrial node cells: numerical modeling of the coupling process.舒张期钙释放控制兔窦房结细胞的搏动频率：耦合过程的数值模拟
Biophys J. 2004 Apr;86(4):2596-605. doi: 10.1016/S0006-3495(04)74314-3.
7
Ionic basis of ischemia-induced bradycardia in the rabbit sinoatrial node.兔窦房结缺血性心动过缓的离子基础。
J Mol Cell Cardiol. 2007 Feb;42(2):315-25. doi: 10.1016/j.yjmcc.2006.10.004. Epub 2006 Nov 13.
8
Comment on "limit cycle oscillations in pacemaker cells".关于“起搏器细胞中的极限环振荡”的评论
IEEE Trans Biomed Eng. 2001 Apr;48(4):499-500. doi: 10.1109/10.915720.
9
Mechanisms of beat-to-beat regulation of cardiac pacemaker cell function by Ca²⁺ cycling dynamics.钙循环动力学对心脏起搏细胞功能的逐搏调节机制。
Biophys J. 2013 Oct 1;105(7):1551-61. doi: 10.1016/j.bpj.2013.08.024.
10
Diastolic release of calcium from the sarcoplasmic reticulum: a potential target for treating triggered arrhythmias and heart failure.
J Am Coll Cardiol. 2009 May 26;53(21):2006-8. doi: 10.1016/j.jacc.2009.02.032.

引用本文的文献

1
Ca(2+) cycling properties are conserved despite bradycardic effects of heart failure in sinoatrial node cells.尽管心力衰竭对窦房结细胞有心动过缓作用，但钙（Ca2+）循环特性仍得以保留。
Front Physiol. 2015 Feb 2;6:18. doi: 10.3389/fphys.2015.00018. eCollection 2015.
2
Functional role of CLC-2 chloride inward rectifier channels in cardiac sinoatrial nodal pacemaker cells.CLC-2 氯离子内向整流通道在心脏窦房结起搏细胞中的功能作用。
J Mol Cell Cardiol. 2009 Jul;47(1):121-32. doi: 10.1016/j.yjmcc.2009.04.008. Epub 2009 Apr 17.
3
Synergism of coupled subsarcolemmal Ca2+ clocks and sarcolemmal voltage clocks confers robust and flexible pacemaker function in a novel pacemaker cell model.
耦合的肌膜下钙时钟和肌膜电压时钟的协同作用在一种新型起搏细胞模型中赋予强大且灵活的起搏器功能。
Am J Physiol Heart Circ Physiol. 2009 Mar;296(3):H594-615. doi: 10.1152/ajpheart.01118.2008. Epub 2009 Jan 9.
4
Generation and escape of local waves from the boundary of uncoupled cardiac tissue.未耦合心脏组织边界处局部波的产生与逸出。
Biophys J. 2008 May 1;94(9):3726-38. doi: 10.1529/biophysj.107.117630. Epub 2008 Jan 22.
5
Normal heart rhythm is initiated and regulated by an intracellular calcium clock within pacemaker cells.正常心律由起搏细胞内的细胞内钙钟启动和调节。
Heart Lung Circ. 2007 Oct;16(5):335-48. doi: 10.1016/j.hlc.2007.07.005. Epub 2007 Sep 10.
6
Cardiac pacemaker cell failure with preserved I(f), I(CaL), and I(Kr): a lesson about pacemaker function learned from ischemia-induced bradycardia.心脏起搏器细胞功能衰竭但I(f)、I(CaL)和I(Kr)保留：从缺血性心动过缓中学到的关于起搏器功能的一课。
J Mol Cell Cardiol. 2007 Feb;42(2):289-94. doi: 10.1016/j.yjmcc.2006.11.009. Epub 2006 Dec 22.