斑秃中Fc受体样3（FCRL3）基因功能性变异c.-169T>C的研究。
Investigation of the functional variant c.-169T > C of the Fc receptor-like 3 (FCRL3) gene in alopecia areata.
作者信息
Schäfer N, Blaumeiser B, Becker T, Freudenberg-Hua Y, Hanneken S, Eigelshoven S, Schmael C, Lambert J, De Weert J, Kruse R, Nöthen M M, Betz R C
机构信息
Institute of Human Genetics, University of Bonn, Bonn, Germany.
出版信息
Int J Immunogenet. 2006 Dec;33(6):393-5. doi: 10.1111/j.1744-313X.2006.00633.x.
A functional variant in the Fc receptor-like 3 (FCRL3) gene has been implicated in susceptibility to autoimmune diseases such as rheumatoid arthritis, systemic lupus erythematosus and autoimmune thyroid disease. Investigating a large case-control sample of patients with alopecia areata (AA), we found no evidence for the involvement of FCRL3 in susceptibility to AA.
Fc受体样3（FCRL3）基因中的一个功能性变体与类风湿性关节炎、系统性红斑狼疮和自身免疫性甲状腺疾病等自身免疫性疾病的易感性有关。在对大量斑秃（AA）患者进行病例对照研究时，我们没有发现FCRL3参与AA易感性的证据。
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