• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

Inflammatory cells are sources of polyamines that induce alveolar macrophage to undergo apoptosis during Pneumocystis pneumonia.

作者信息

Liao Chung-Ping, Lasbury Mark E, Wang Shao-Hung, Zhang Chen, Durant Pamela J, Tschang Dennis, Lee Chao-Hung

机构信息

Department of Pathology and Laboratory Medicine, Indiana University School of Medicine, 1120 South Drive, Fesler Hall Rm 419, Indianapolis, Indiana 46202, USA.

出版信息

J Eukaryot Microbiol. 2006;53 Suppl 1:S134-5. doi: 10.1111/j.1550-7408.2006.00203.x.

DOI:10.1111/j.1550-7408.2006.00203.x
PMID:17169031
Abstract
摘要

相似文献

1
Inflammatory cells are sources of polyamines that induce alveolar macrophage to undergo apoptosis during Pneumocystis pneumonia.炎症细胞是多胺的来源，多胺在肺孢子菌肺炎期间可诱导肺泡巨噬细胞发生凋亡。
J Eukaryot Microbiol. 2006;53 Suppl 1:S134-5. doi: 10.1111/j.1550-7408.2006.00203.x.
2
Alterations in surfactant protein A form and clearance during Pneumocystis pneumonia.卡氏肺孢子虫肺炎期间表面活性物质蛋白A的形式和清除率的改变。
J Eukaryot Microbiol. 2006;53 Suppl 1:S119-21. doi: 10.1111/j.1550-7408.2006.00197.x.
3
Expression and activation of complement protein and alveolar damage during Pneumocystis pneumonia.卡氏肺孢子虫肺炎期间补体蛋白的表达与激活及肺泡损伤
J Eukaryot Microbiol. 2006;53 Suppl 1:S136-7. doi: 10.1111/j.1550-7408.2006.00204.x.
4
Transforming growth factor-beta activation and signaling in the alveolar environment during Pneumocystis pneumonia.卡氏肺孢子虫肺炎期间肺泡环境中转化生长因子-β的激活与信号传导
J Eukaryot Microbiol. 2006;53 Suppl 1:S127-9. doi: 10.1111/j.1550-7408.2006.00200.x.
5
GM-CSF expression in the lung during Pneumocystis pneumonia.卡氏肺孢子虫肺炎时肺内粒细胞-巨噬细胞集落刺激因子的表达
J Eukaryot Microbiol. 2006;53 Suppl 1:S124-6. doi: 10.1111/j.1550-7408.2006.00199.x.
6
Pneumocystis mediates overexpression of antizyme inhibitor resulting in increased polyamine levels and apoptosis in alveolar macrophages.肺孢子菌介导抗酶抑制剂的过表达，导致肺泡巨噬细胞中多胺水平升高和细胞凋亡。
J Biol Chem. 2009 Mar 20;284(12):8174-84. doi: 10.1074/jbc.M805787200. Epub 2009 Jan 20.
7
Survival pathway signal transduction is reduced in alveolar macrophages during Pneumocystis pneumonia.在肺孢子菌肺炎期间，肺泡巨噬细胞中的生存途径信号转导减少。
J Eukaryot Microbiol. 2006;53 Suppl 1:S130-1. doi: 10.1111/j.1550-7408.2006.00201.x.
8
Polyamine-mediated apoptosis of alveolar macrophages during Pneumocystis pneumonia.多胺介导的肺孢子菌肺炎期间肺泡巨噬细胞凋亡
J Biol Chem. 2007 Apr 13;282(15):11009-20. doi: 10.1074/jbc.M611686200. Epub 2007 Feb 21.
9
Toll-like receptor 2 knockout reduces lung inflammation during Pneumocystis pneumonia but has no effect on phagocytosis of Pneumocystis organisms by alveolar macrophages.Toll样受体2基因敲除可减轻肺孢子菌肺炎期间的肺部炎症，但对肺泡巨噬细胞吞噬肺孢子菌生物体没有影响。
J Eukaryot Microbiol. 2006;53 Suppl 1:S132-3. doi: 10.1111/j.1550-7408.2006.00202.x.
10
Caspase-9 as a target for Pneumocystis pneumonia therapy.半胱天冬酶-9作为卡氏肺孢子虫肺炎治疗的靶点。
J Eukaryot Microbiol. 2006;53 Suppl 1:S138-9. doi: 10.1111/j.1550-7408.2006.00205.x.

引用本文的文献

1
Natural polyamine inhibits mouse skin inflammation and macrophage activation.天然多胺抑制小鼠皮肤炎症和巨噬细胞活化。
Inflamm Res. 2013 Jul;62(7):681-8. doi: 10.1007/s00011-013-0620-5. Epub 2013 Apr 19.