Lutsevich E E
Vestn Oftalmol. 2006 Nov-Dec;122(6):6-9.
The paper deals with the basic pathogenetic aspects of development of orbital hernias--the factors of a tarsoorbital fascial change in the presence of the hereditary syndrome of connective tissue hyperplasticity and elevated intraorbital pressure, which affects the volume of orbital fat. The possibilities of a differential diagnosis of orbital hernias and eyelid edemas are considered. There is a biomechanical association of orbital hernias with acquired age-related enophthalmos. The examples of impairments in the tolerance of the optic nerve and in the development of optic neuropathy in enophthalmos are considered. The fact that there may be tarsoorbital fascial lesions, followed by the development of orbital hernias after parabulbar injections is indicated. The author proposes an operation dealing with the reposition of orbital hernias instead of their resection during blepharoplastic interventions.
本文探讨眼眶疝形成的基本发病机制——在结缔组织增生性遗传综合征和眶内压升高的情况下睑眶筋膜变化的因素,这会影响眶脂肪体积。文中考虑了眼眶疝与眼睑水肿的鉴别诊断可能性。眼眶疝与后天性年龄相关性眼球内陷存在生物力学关联。文中还考虑了眼球内陷时视神经耐受性受损和视神经病变发展的实例。指出在球后注射后可能会出现睑眶筋膜病变,随后发展为眼眶疝。作者提议在眼睑整形手术中对眼眶疝进行复位而非切除的手术方法。