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三只猫的猫炎性息肉的非典型表现。

Atypical manifestations of feline inflammatory polyps in three cats.

作者信息

MacPhail Catriona M, Innocenti Christi M, Kudnig Simon T, Veir Julia K, Lappin Michael R

机构信息

Department of Clinical Sciences, College of Veterinary Medicine and Biomedical Sciences, Colorado State University, Fort Collins, CO 80523, USA.

出版信息

J Feline Med Surg. 2007 Jun;9(3):219-25. doi: 10.1016/j.jfms.2006.11.004. Epub 2007 Jan 22.

Abstract

Inflammatory polyps of the feline middle ear and nasopharynx are non-neoplastic masses that are presumed to originate from the epithelial lining of the tympanic bulla or Eustachian tube. The exact origin and cause are unknown, however, it is thought that inflammatory polyps arise as a result of a prolonged inflammatory process. It is unclear whether this inflammation initiates or potentiates the development and growth of inflammatory polyps. Cats with inflammatory polyps typically present with either signs of otitis externa and otitis media or with signs consistent with upper airway obstruction. Traditional diagnostics involve imaging of the tympanic bulla either with skull radiographs or computed topography (CT). Treatment consists of traction and avulsion of the polyp with or without ventral bulla osteotomy (VBO) to remove the epithelial lining of the tympanic bulla. The three cases described here are unusual manifestations or presentations of feline inflammatory polyps that address the following issues: (1) concurrent otic and nasopharyngeal polyps, (2) potential association with chronic viral infection, (3) polyp development in the contralateral middle ear, (4) CT appearance of the skull following VBO, and (5) development of secondary pulmonary hypertension.

摘要

猫中耳和鼻咽部的炎性息肉是非肿瘤性肿块,推测起源于鼓泡或咽鼓管的上皮衬里。确切的起源和病因尚不清楚,然而,人们认为炎性息肉是长期炎症过程的结果。目前尚不清楚这种炎症是引发还是促进了炎性息肉的发生和生长。患有炎性息肉的猫通常表现为外耳道炎和中耳炎的症状,或表现为与上呼吸道阻塞一致的症状。传统诊断方法包括通过颅骨X线片或计算机断层扫描(CT)对鼓泡进行成像。治疗方法包括用或不用腹侧鼓泡截骨术(VBO)牵引和撕脱息肉,以去除鼓泡的上皮衬里。这里描述的三个病例是猫炎性息肉的不寻常表现或呈现,涉及以下问题:(1)同时存在耳部和鼻咽部息肉,(2)与慢性病毒感染的潜在关联,(3)对侧中耳息肉发展,(4)VBO后颅骨的CT表现,以及(5)继发性肺动脉高压的发展。

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