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预激综合征伴宽QRS波群心动过速。

Wolf-parkinson-white syndrome presented with broad QRS complex tachycardia.

作者信息

Manurung Daulat, Yamin M

机构信息

Division of Cardiology, Department of Internal Medicine, Faculty of Medicine, University of Indonesia-Cipto Mangunkusumo Hospital, Jakarta.

出版信息

Acta Med Indones. 2007 Jan-Mar;39(1):33-5.

Abstract

Broad QRS complex tachycardia is tachycardia with widened QRS complex more than 12 s and caused by various mechanisms, either supraventricular or ventricular. It is important to differentiate between ventricular and supraventricular because it will determine treatment and prognosis of patients. We report a case which was referred to us and first diagnosed as ventricular tachycardia but happened to be atrial fibrillation with RBBB. On ECG examination we found irregular broad complex of tachycardia, RBBB, extreme right axis and heart rate 170-180 beat/minute. Intravenous bolus of 300 mg amiodarone was administered within 30 minutes and continued with 900 mg/24 hours. During administration of amiodarone, heart rhythm was converted to sinus rhythm with short PR interval (0.09 s), left axis deviation, and positive delta wave at lead V1. The final diagnosis of wolf-parkinson-white (WPW) syndrome was then confirmed.

摘要

宽QRS波群心动过速是指QRS波群增宽超过12秒的心动过速,由多种机制引起,包括室上性或室性机制。区分室性和室上性心动过速很重要,因为这将决定患者的治疗和预后。我们报告一例转诊至我院的病例,最初被诊断为室性心动过速,但实际上是伴有右束支传导阻滞的心房颤动。心电图检查发现心动过速的宽大复合波不规则、右束支传导阻滞、极度电轴右偏且心率为170 - 180次/分钟。在30分钟内静脉推注300毫克胺碘酮,并持续以900毫克/24小时给药。在胺碘酮给药期间,心律转为窦性心律,PR间期缩短(0.09秒),电轴左偏,V1导联出现正向δ波。随后确诊为预激综合征。

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