Wienecke M M, Case C L, Gillette P C
Medical University of South Carolina, Children's Heart Center, Charleston 29425.
Clin Cardiol. 1992 Feb;15(2):114-6. doi: 10.1002/clc.4960150212.
Adenosine's ability to terminate atrioventricular (AV) re-entrant supraventricular tachycardia is well documented. Typically, termination occurs as a consequence of transient conduction block in the atrioventricular node, a tissue with decremental qualities. However, the atrioventricular node is not always the site of action when adenosine is used on the re-entrant types of long RP' tachycardias. These tachycardias are, in part, characterized by the decremental qualities of the retrograde limb of the tachycardia circuit, which, in turn, are typically exemplified by retrograde Wenckebach during ventricular (VVI) pacing during intracardiac electrophysiology studies. This case report involves adenosine's ability to block conduction in the retrograde limb of the permanent form of junctional reciprocating tachycardia to provide further evidence as to the AV "nodelike" decremental qualities of this limb.
腺苷终止房室(AV)折返性室上性心动过速的能力已有充分文献记载。通常,终止是由于房室结(一种具有递减特性的组织)出现短暂传导阻滞所致。然而,在用于折返型长RP'心动过速时,腺苷的作用部位并不总是在房室结。这些心动过速部分特征为心动过速环路逆行支具有递减特性,这在心脏电生理研究中,心室(VVI)起搏时的逆行文氏现象中通常有所体现。本病例报告涉及腺苷阻断永久性交界性折返性心动过速逆行支传导的能力,以进一步证明该支具有房室“结样”递减特性。