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连续脊麻后持续性骶神经根功能缺损

Persistent sacral nerve root deficits after continuous spinal anaesthesia.

作者信息

Schell R M, Brauer F S, Cole D J, Applegate R L

机构信息

Department of Anesthesiology, Loma Linda University, California 92350.

出版信息

Can J Anaesth. 1991 Oct;38(7):908-11. doi: 10.1007/BF03036972.

Abstract

Neurological deficits following spinal anaesthesia are rare. We report two cases of persistent sacral nerve root deficits after continuous spinal anaesthesia (CSA) performed with hyperbaric lidocaine through a lumbar microcatheter. In both cases the dose of 5% lidocaine (5.7 and 4.3 ml) was greater than usual. In the immediate postoperative period the constellation of neurological deficits included perianal hypaesthesia, lower extremity paresis, urinary retention, and difficult defaecation. Both patients have residual perianal hypaesthesia and difficult defaecation. In these cases, the high-dose requirements of local anaesthetic via microcatheter CSA with focal sensory block suggests nonuniform distribution of the hyperbaric lidocaine. Microcatheter CSA may convey a unique risk of maldistribution of the local anaesthetic solution and local neurotoxicity.

摘要

脊髓麻醉后出现神经功能缺损的情况较为罕见。我们报告了两例通过腰椎微导管使用重比重利多卡因进行连续脊髓麻醉(CSA)后出现持续性骶神经根功能缺损的病例。在这两例中,5%利多卡因的剂量(分别为5.7毫升和4.3毫升)均高于常规剂量。术后即刻,神经功能缺损的表现包括肛周感觉减退、下肢轻瘫、尿潴留和排便困难。两名患者均遗留肛周感觉减退和排便困难。在这些病例中,通过微导管CSA给予高剂量局麻药并产生局部感觉阻滞,提示重比重利多卡因分布不均。微导管CSA可能会带来局麻药液分布不均和局部神经毒性的独特风险。

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