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对关键肌筋膜触发点进行干针疗法可能会降低卫星型肌触发点的兴奋性。

Dry needling to a key myofascial trigger point may reduce the irritability of satellite MTrPs.

作者信息

Hsieh Yueh-Ling, Kao Mu-Jung, Kuan Ta-Shen, Chen Shu-Min, Chen Jo-Tong, Hong Chang-Zern

机构信息

Department of Physical Therapy, Hungkuang University, Salu, Taichung, Taiwan.

出版信息

Am J Phys Med Rehabil. 2007 May;86(5):397-403. doi: 10.1097/PHM.0b013e31804a554d.

Abstract

OBJECTIVE

To investigate the changes in pressure pain threshold of the secondary (satellite) myofascial trigger points (MTrPs) after dry needling of a primary (key) active MTrP.

DESIGN

Single blinded within-subject design, with the same subjects serving as their own controls (randomized). Fourteen patients with bilateral shoulder pain and active MTrPs in bilateral infraspinatus muscles were involved. An MTrP in the infraspinatus muscle on a randomly selected side was dry needled, and the MTrP on the contralateral side was not (control). Shoulder pain intensity, range of motion (ROM) of shoulder internal rotation, and pressure pain threshold of the MTrPs in the infraspinatus, anterior deltoid, and extensor carpi radialis longus muscles were measured in both sides before and immediately after dry needling.

RESULTS

Both active and passive ROM of shoulder internal rotation, and the pressure pain threshold of MTrPs on the treated side, were significantly increased (P < 0.01), and the pain intensity of the treated shoulder was significantly reduced (P < 0.001) after dry needling. However, there were no significant changes in all parameters in the control (untreated) side. Percent changes in the data after needling were also analyzed. For every parameter, the percent change was significantly higher in the treated side than in the control side.

CONCLUSIONS

This study provides evidence that dry needle-evoked inactivation of a primary (key) MTrP inhibits the activity in satellite MTrPs situated in its zone of pain referral. This supports the concept that activity in a primary MTrP leads to the development of activity in satellite MTrPs and the suggested spinal cord mechanism responsible for this phenomenon.

摘要

目的

研究原发性(关键)活动性肌筋膜触发点(MTrP)进行干针治疗后,继发性(卫星)MTrP的压痛阈值变化。

设计

单盲自身对照设计,同一受试者作为自身对照(随机分组)。纳入14例双侧肩部疼痛且双侧冈下肌存在活动性MTrP的患者。随机选择一侧的冈下肌MTrP进行干针治疗,对侧MTrP不进行治疗(对照)。在干针治疗前及治疗后即刻,测量双侧肩部疼痛强度、肩部内旋活动范围(ROM)以及冈下肌、三角肌前部和桡侧腕长伸肌MTrP的压痛阈值。

结果

干针治疗后,肩部内旋的主动和被动ROM以及治疗侧MTrP的压痛阈值均显著增加(P < 0.01),治疗肩部的疼痛强度显著降低(P < 0.001)。然而,对照(未治疗)侧的所有参数均无显著变化。还分析了针刺后数据的百分比变化。对于每个参数,治疗侧的百分比变化均显著高于对照侧。

结论

本研究提供了证据,表明原发性(关键)MTrP经干针诱发失活可抑制位于其疼痛牵涉区内的卫星MTrP的活性。这支持了原发性MTrP的活性会导致卫星MTrP活性增加的概念,以及为此现象所提出的脊髓机制。

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