Lautebach S, Funk J, Reinhard T, Pache M
Universitäts-Augenklinik Freiburg.
Klin Monbl Augenheilkd. 2007 May;224(5):438-40. doi: 10.1055/s-2007-963095.
A steroid-induced glaucoma may develop after bilateral laser in situ keratomileusis (LASIK) with normal intraocular pressure in applanation tonometry.
We present the case of a 32-year-old patient who underwent bilateral LASIK for myopia. Postoperatively, a steroid-induced glaucoma developed. After the steroid therapy was stopped applanation tonometry showed normal values. A slight corneal opacity was interpreted as a keratokonjunctivitis sicca because of occupational noxa. One year after LASIK, the patient presented with high intraocular pressure (IOP), maximally excavated optic nerve head and extensive visual fields defect in both eyes.
Elevated IOP after LASIK can lead to fluid accumulation in the interface. In this case applanation tonometry can underestimate the intraocular pressure. Even when steroid therapy is stopped, the elevated pressure can persist. This complication after LASIK is very rare and can cause severe damage if not diagnosed.
在双眼准分子原位角膜磨镶术(LASIK)后,眼压在压平眼压计测量下正常时,仍可能发生类固醇性青光眼。
我们报告一例32岁近视患者接受双眼LASIK手术的病例。术后发生了类固醇性青光眼。停用类固醇治疗后,压平眼压计显示眼压值正常。因职业性有害因素,轻微的角膜混浊被解释为干眼症。LASIK术后一年,患者双眼出现高眼压、视神经乳头最大程度凹陷及广泛的视野缺损。
LASIK术后眼压升高可导致界面处积液。在此病例中,压平眼压计可能低估眼压。即使停用类固醇治疗,升高的眼压仍可能持续。LASIK术后的这种并发症非常罕见,如果未被诊断,可能会造成严重损害。