Gerard Elizabeth, Frontera Jennifer A, Wright Clinton B
Division of Stroke and Critical Care Neurology, Neurological Institute, College of Physicians and Surgeons, Columbia University, 710 West 168th Street, New York, NY 10032, USA.
Neurocrit Care. 2007;7(3):257-9. doi: 10.1007/s12028-007-0057-1.
Cerebral arterial vasospasm following aneurysmal subarachnoid hemorrhage (SAH) is an important cause of delayed neurologic deterioration. Vasospasm following isolated intraventricular hemorrhage (IVH) is less common. Accepted predictors of vasospasm following SAH include poor Hunt-Hess grade, elevated transcranial Doppler velocities, and the thickness of cisternal blood on neuroimaging [1, 2]. The role of intraventricular hemorrhage in vasospasm is more controversial.
Case report and review of the literature.
A 41-year-old woman developed symptomatic delayed vasospasm 10 days following isolated IVH due to the rupture of an arteriovenous malformation (AVM).
Intraventricular hemorrhage can independently cause significant delayed vasospasm. Possible mechanisms are described.
动脉瘤性蛛网膜下腔出血(SAH)后的脑动脉血管痉挛是导致延迟性神经功能恶化的重要原因。孤立性脑室内出血(IVH)后的血管痉挛较少见。SAH后血管痉挛公认的预测因素包括Hunt-Hess分级差、经颅多普勒速度升高以及神经影像学上脑池内血液厚度[1,2]。脑室内出血在血管痉挛中的作用更具争议性。
病例报告及文献复习。
一名41岁女性因动静脉畸形(AVM)破裂导致孤立性IVH后10天出现有症状的延迟性血管痉挛。
脑室内出血可独立导致显著的延迟性血管痉挛。文中描述了可能的机制。