Shinde Rituparna, Shinde Suresh, Makhale Chandrashekhar, Grant Purvez, Sathe Sunil, Durairaj Manuel, Lokhandwala Yash, Di Diego Jose, Antzelevitch Charles
Department of Cardiology, Grant Medical Foundation, Ruby Hall Clinic, Pune, Maharashtra 411001, India.
Pacing Clin Electrophysiol. 2007 Jun;30(6):817-9. doi: 10.1111/j.1540-8159.2007.00760.x.
The "J wave" (also referred to as "the Osborn wave,""the J deflection," or "the camel's hump") is a distinctive deflection occurring at the QRS-ST junction. In 1953, Dr. John Osborn described the "J wave" as an "injury current" resulting in ventricular fibrillation during experimental hypothermia. Although "J Wave" is supposed to be pathognomonic of hypothermia, it is seen in a host of other conditions such as hypercalcemia, brain injury, subarachnoid hemorrhage, cardiopulmonary arrest from over sedation, the Brugada syndrome, vasospastic angina, and idiopathic ventricular fibrillation. However, there is paucity of literature data as regards to ischemic etiology of "J Wave." In this article, we present a case where "J waves" were probably induced by ischemia. We also discuss the mechanism of ischemia-induced "J wave" accentuation and its prognostic implications.
“J波”（也称为“奥斯本波”“J波偏移”或“驼峰”）是在QRS波群与ST段交界处出现的一种特征性偏移。1953年，约翰·奥斯本博士将“J波”描述为实验性低温期间导致心室颤动的“损伤电流”。尽管“J波”被认为是低温的特征性表现，但它也见于许多其他情况，如高钙血症、脑损伤、蛛网膜下腔出血、过度镇静导致的心肺骤停、布加综合征、变异性心绞痛和特发性心室颤动。然而，关于“J波”的缺血性病因，文献资料较少。在本文中，我们报告了一例“J波”可能由缺血诱发的病例。我们还讨论了缺血诱发“J波”增强的机制及其预后意义。
