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Point:Counterpoint "Release of an endothelium-derived vasoconstrictor and RhoA/Rho kinase-mediated calcium sensitization of smooth muscle cell contraction are/are not the main effectors for full and sustained hypoxic pulmonary vasoconstriction".

作者信息

Yuan Jason X-J

机构信息

University of California, San Diego, USA.

出版信息

J Appl Physiol (1985). 2007 May;102(5):2078; discussion 2080. doi: 10.1152/japplphysiol.01435.2006.

DOI:10.1152/japplphysiol.01435.2006
PMID:17549820
Abstract
摘要

相似文献

1
Point:Counterpoint "Release of an endothelium-derived vasoconstrictor and RhoA/Rho kinase-mediated calcium sensitization of smooth muscle cell contraction are/are not the main effectors for full and sustained hypoxic pulmonary vasoconstriction".正方观点对反方观点：“内皮源性血管收缩因子的释放以及RhoA/Rho激酶介导的平滑肌细胞收缩钙敏化是/不是完全且持续的低氧性肺血管收缩的主要效应器”
J Appl Physiol (1985). 2007 May;102(5):2078; discussion 2080. doi: 10.1152/japplphysiol.01435.2006.
2
Point:Counterpoint: "Release of an endothelium-derived vasoconstrictor and RhoA/Rho kinase-mediated calcium sensitization of smooth muscle cell contraction are/are not the main effectors for full and sustained hypoxic pulmonary vasoconstriction".观点：对立观点：“内皮源性血管收缩剂的释放以及RhoA/Rho激酶介导的平滑肌细胞收缩钙敏化是/不是完全且持续的低氧性肺血管收缩的主要效应器” 。
J Appl Physiol (1985). 2007 May;102(5):2077; discussion 2080. doi: 10.1152/japplphysiol.01435.2006.
3
Point:Counterpoint "Release of an endothelium-derived vasoconstrictor and RhoA/Rho kinase-mediated calcium sensitization of smooth muscle cell contraction are/are not the main effectors for full and sustained hypoxic pulmonary vasoconstriction".正方观点对反方观点：“内皮源性血管收缩因子的释放以及RhoA/Rho激酶介导的平滑肌细胞收缩钙敏化是/不是完全且持续的低氧性肺血管收缩的主要效应器”
J Appl Physiol (1985). 2007 May;102(5):2078-9; discussion 2080. doi: 10.1152/japplphysiol.01435.2006.
4
Point:Counterpoint "Release of an endothelium-derived vasoconstrictor and RhoA/Rho kinase-mediated calcium sensitization of smooth muscle cell contraction are/are not the main effectors for full and sustained hypoxic pulmonary vasoconstriction".正方观点：反方观点 “内皮源性血管收缩因子的释放以及RhoA/Rho激酶介导的平滑肌细胞收缩钙敏化是/不是完全性和持续性低氧性肺血管收缩的主要效应器”。
J Appl Physiol (1985). 2007 May;102(5):2077-8; discussion 2080. doi: 10.1152/japplphysiol.01435.2006.
5
Point: release of an endothelium-derived vasoconstrictor and RhoA/Rho kinase-mediated calcium sensitization of smooth muscle cell contraction are/are not the main effectors for full and sustained hypoxic pulmonary vasoconstriction.观点：内皮源性血管收缩因子的释放以及RhoA/Rho激酶介导的平滑肌细胞收缩的钙敏化作用是/不是完全且持续的低氧性肺血管收缩的主要效应机制。
J Appl Physiol (1985). 2007 May;102(5):2071-2; discussion 2075-6. doi: 10.1152/japplphysiol.01258.2006. Epub 2006 Nov 9.
6
Point:Counterpoint "Release of an endothelium-derived vasoconstrictor and RhoA/Rho kinase-mediated calcium sensitization of smooth muscle cell contraction are/are not the main effectors for full and sustained hypoxic pulmonary vasoconstriction".观点：对立观点“内皮源性血管收缩剂的释放以及RhoA/Rho激酶介导的平滑肌细胞收缩钙敏化是/不是完全且持续的低氧性肺血管收缩的主要效应器”
J Appl Physiol (1985). 2007 May;102(5):2077; discussion 2080. doi: 10.1152/japplphysiol.01435.2006.
7
Counterpoint: release of an endothelium-derived vasoconstrictor and RhoA/Rho kinase-mediated calcium sensitization of smooth muscle cell contraction are not the main effectors for full and sustained HPV.相反观点：内皮细胞衍生的血管收缩剂的释放以及RhoA/Rho激酶介导的平滑肌细胞收缩的钙敏化并非完全和持续性HPV的主要效应器。
J Appl Physiol (1985). 2007 May;102(5):2072-5; discussion 2075, 2080. doi: 10.1152/japplphysiol.01258a.2006.
8
Reactive oxygen species mediate RhoA/Rho kinase-induced Ca2+ sensitization in pulmonary vascular smooth muscle following chronic hypoxia.活性氧介导慢性低氧后肺血管平滑肌中RhoA/Rho激酶诱导的Ca2+致敏。
Am J Physiol Lung Cell Mol Physiol. 2008 Sep;295(3):L515-29. doi: 10.1152/ajplung.00355.2007. Epub 2008 Jul 11.
9
Sildenafil prevents change in RhoA expression induced by chronic hypoxia in rat pulmonary artery.西地那非可预防大鼠肺动脉慢性缺氧诱导的RhoA表达变化。
Circ Res. 2003 Oct 3;93(7):630-7. doi: 10.1161/01.RES.0000093220.90027.D9. Epub 2003 Aug 28.
10
Rho-kinase activation is involved in hypoxia-induced pulmonary vasoconstriction.Rho激酶激活参与缺氧诱导的肺血管收缩。
Am J Respir Cell Mol Biol. 2001 Nov;25(5):628-35. doi: 10.1165/ajrcmb.25.5.4461.