准分子原位角膜磨镶术后因类固醇诱导的高眼压导致的板层间瓣水肿。
Interlamellar flap edema due to steroid-induced ocular hypertension after laser in situ keratomileusis.
作者信息
Miyai Takashi, Yonemura Takaharu, Nejima Ryohei, Otani Shinichiro, Miyata Kazunori, Amano Shiro
机构信息
Meiwakai Medical Foundation, Miyata Eye Hospital, Miyakonojo, Miyazaki, Japan.
出版信息
Jpn J Ophthalmol. 2007 May-Jun;51(3):228-30. doi: 10.1007/s10384-006-0441-y. Epub 2007 Jun 7.
BACKGROUND
Several recent studies have reported post-laser in situ keratomileusis (LASIK) complications related to a steroid-induced increase in intraocular pressure, including interface fluid and elevated intraocular pressure-induced interlamellar stromal keratitis.
CASES
We examined two cases of interlamellar flap edema due to steroid-induced ocular hypertension after uneventful laser in situ keratomileusis.
OBSERVATIONS
Oral acetazolamide and discontinuance of topical steroids were effective for treating interlamellar stromal edema in both cases.
CONCLUSION
A rise in intraocular pressure should be considered a cause of interlamellar stromal edema after LASIK.
背景
最近的几项研究报告了准分子原位角膜磨镶术（LASIK）后与类固醇诱导的眼压升高相关的并发症，包括界面积液和眼压升高引起的板层间基质角膜炎。
病例
我们检查了两例在LASIK手术顺利完成后因类固醇诱导的高眼压导致的板层间瓣水肿病例。
观察结果
口服乙酰唑胺和停用局部类固醇对两例病例的板层间基质水肿均有效。
结论
眼压升高应被视为LASIK术后板层间基质水肿的一个原因。
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