Galvis Virgilio, Tello Alejandro, Revelo Mario Leandro, Valarezo Paul
Department of Ophthalmology, Centro Oftalmologico Virgilio Galvis, Floridablanca, Santander, Colombia.
BMJ Case Rep. 2012 Oct 30;2012:bcr2012007328. doi: 10.1136/bcr-2012-007328.
A 33-year-old man who underwent uneventful laser in situ keratomileusis (LASIK) developed pressure-induced stromal edema resulting in an interface haze in both eyes and a pocket of fluid under the flap of the right eye 10 days after surgery, while receiving topical fluorometholone. Intraocular pressure by applanation tonometry was 16 mm Hg in his right eye (erroneous result due to the fluid in the interface) and 34 mm Hg in his left eye. After discontinuation of steroids and addition of ocular hypotensive medication, interface fluid collection disappeared in his right eye. Visual acuity improved and haze diminished in both eyes. This case illustrates that in the same patient a post-LASIK edema induced syndrome may be present with or without fluid in the interface, suggesting that both clinical pictures could be manifestations of a broad spectrum of the same condition. We suggest a new name for this non-inflammatory disorder: post-LASIK edema-induced keratopathy (PLEK).
一名33岁男性接受了顺利的准分子原位角膜磨镶术（LASIK），术后10天，在使用局部氟米龙时，双眼出现压力诱导的基质水肿，导致界面混浊，右眼瓣下有液体积聚。应用压平眼压计测量，右眼眼压为16 mmHg（因界面有液体导致结果错误），左眼眼压为34 mmHg。停用类固醇并加用降眼压药物后，右眼界面液体积聚消失。双眼视力提高，混浊减轻。该病例表明，同一患者的LASIK术后水肿诱导综合征可能伴有或不伴有界面积液，提示这两种临床表现可能是同一疾病广泛谱的表现。我们建议为这种非炎症性疾病命名为：LASIK术后水肿诱导性角膜病变（PLEK）。
