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[生长发育期创伤后轴向排列不齐的原理与发病机制]

[Principles and pathogenesis of post-traumatic axial malalignment in the growth years].

作者信息

Hefti F, von Laer L, Morscher E

机构信息

Orthopädische und Kinderchirurgische, Universitätsklinik Basel.

出版信息

Orthopade. 1991 Nov;20(6):324-30.

PMID:1758696
Abstract

Deviations of the axis or leg-length discrepancies after fractures in children and adolescents can be due to growth disturbances or can be the result of incomplete reduction of the fracture. We distinguish between four types of growth disturbances. In type I, the overall growth activity of the cartilage is increased; growth is then enhanced, which results in the affected bone being too long without deviation; this usually occurs after fractures of the metaphysis or diaphysis. In type II, activity, the epiphyseal cartilage is severely impaired or completely arrested. The direction of growth is unchanged. This results in shortening of the bone, usually due to severe damage to the germination zone of the growth cartilage after destruction of the vessels or infection. In type III, growth of the epiphyseal plate is partially stimulated. The consequence of this disturbance is deviation of the axis with overgrowth (this is in fractures of the proximal tibia). Type IV is characterized by an asymmetric arrest of growth. This results in deviation of the axis and shortening. The cause of such growth arrest can be epiphyseolysis or epiphyseal fracture. The defect in growth cartilage heals with a bone bridge. This is a very serious kind of growth disturbance, and it occurs in only 1% of all fractures before skeletal maturity. Correction after incomplete reduction of fractures during growth can be direct or indirect and specific or nonspecific. Direct corrections occur in combination with fracture healing; indirect corrections occur with physiological changes of the growing skeleton without association with the healing process.(ABSTRACT TRUNCATED AT 250 WORDS)

摘要

儿童和青少年骨折后出现的轴线偏差或腿长差异,可能是由于生长紊乱,也可能是骨折复位不完全所致。我们将生长紊乱分为四种类型。I型中,软骨的整体生长活性增加;随后生长加速,导致患骨过长但无偏差;这通常发生在干骺端或骨干骨折后。II型中,骨骺软骨活性严重受损或完全停滞。生长方向不变。这导致骨缩短,通常是由于血管破坏或感染后生长软骨生发区严重受损。III型中,骨骺板的生长部分受到刺激。这种紊乱的后果是轴线偏差并伴有过度生长(这见于胫骨近端骨折)。IV型的特征是生长不对称停滞。这导致轴线偏差和缩短。这种生长停滞的原因可能是骨骺分离或骨骺骨折。生长软骨的缺损通过骨桥愈合。这是一种非常严重的生长紊乱,在骨骼成熟前的所有骨折中仅占1%。生长期间骨折复位不完全后的矫正可分为直接或间接、特异性或非特异性。直接矫正与骨折愈合同时发生;间接矫正随着生长中骨骼的生理变化而发生,与愈合过程无关。(摘要截取自250字)

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