Okumura Yasuo, Watanabe Ichiro, Ohkubo Kimie, Saito Satoshi
Division of Cardiology, Department of Medicine, Nihon University School of Medicine, Tokyo, Japan.
Circ J. 2007 Jul;71(7):1164-8. doi: 10.1253/circj.71.1164.
A 35-year-old man was referred to Nihon University Hospital because of repetitive ventricular tachycardia (VT) at 180-200 beats/min. QRS morphology of the VT was right bundle branch block with a northwest axis. Transthoracic echocardiography showed hypertrophic cardiomyopathy. Coronary angiography was normal and left ventriculography showed neither obstruction in the left ventricle (LV) nor any pressure gradients within the LV or between the LV and aorta. Hemodynamic deterioration occurred during VT. Intracardiac mapping showed that the VT originated from the posteroseptal portion of the LV near the apex and Purkinje potentials that preceded the onset of the QRS complex by 58-70 ms were documented. Radiofrequency ablation at these sites terminated the VT, which has not recurred for 25 months.
一名35岁男性因反复出现心率为180 - 200次/分钟的室性心动过速(VT)被转诊至日本大学医院。VT的QRS形态为右束支传导阻滞伴电轴西北偏。经胸超声心动图显示肥厚型心肌病。冠状动脉造影正常,左心室造影显示左心室(LV)无梗阻,LV内及LV与主动脉之间均无压力阶差。VT发作期间出现血流动力学恶化。心内标测显示VT起源于LV近心尖的后间隔部分,并记录到QRS波群起始前58 - 70毫秒的浦肯野电位。在这些部位进行射频消融终止了VT,此后25个月未再复发。