Gavriysky V S
Department of Physiology and Biochemistry, National Sports Academy, Bulgaria.
Doc Ophthalmol. 1991;77(3):213-24. doi: 10.1007/BF00161369.
We studied the direct and aftereffects of twofold hyperventilation (HV) on pattern reversing VEPs and pupillograms (PGs) of 19 healthy volunteers. The VEP-N80 and P100 latencies increased during HV. Both peak times were maintained for a longer period, up to 20 minutes after HV-2 ended. In addition, the PG-latency time during HV and the PG-construction time during and after HV were increased. The results indicated a temporary delay of neural afferent transmission in the visual system during and after HV. A similar delay of the nervous transmission appeared in the efferent part of the system regulating the pupillary movements after HV ended. The observed changes of the VEP and PG parameters most probably resulted from the hypocapnia cased by HV and its effect on the brain vessels, although other explanations for the changes of the VEP- and PG-parameters may have been possible.
我们研究了双重过度通气（HV）对19名健康志愿者图形翻转视觉诱发电位（VEP）和瞳孔图（PG）的直接影响及后续效应。HV期间，VEP-N80和P100潜伏期延长。两个峰值时间维持较长时间，直至HV-2结束后20分钟。此外，HV期间的PG潜伏期时间以及HV期间和之后的PG构建时间均增加。结果表明，HV期间及之后视觉系统中神经传入传递出现暂时延迟。HV结束后，在调节瞳孔运动系统的传出部分也出现了类似的神经传递延迟。VEP和PG参数的观察到的变化很可能是由HV引起的低碳酸血症及其对脑血管的影响导致的，尽管对于VEP和PG参数的变化可能还有其他解释。
