Copper deficiency, lead, and paraoxonase.
作者信息
Klevay Leslie M
出版信息
Environ Health Perspect. 2007 Jul;115(7):A341-2; author reply A342. doi: 10.1289/ehp.10151.
Abstract
摘要
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Copper deficiency, lead, and paraoxonase.铜缺乏、铅与对氧磷酶。
Environ Health Perspect. 2007 Jul;115(7):A341-2; author reply A342. doi: 10.1289/ehp.10151.
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本文引用的文献
1
How dietary deficiency, genes and a toxin can cooperate to produce arteriosclerosis and ischemic heart disease.饮食缺乏、基因和一种毒素如何协同作用导致动脉硬化和缺血性心脏病。
Cell Mol Biol (Noisy-le-grand). 2006 Dec 31;52(5):11-5.
2
A long and winding road: defining the biological role and clinical importance of paraoxonases.一条漫长而曲折的道路：界定对氧磷酶的生物学作用和临床重要性
Clin Chem Lab Med. 2006;44(9):1052-9. doi: 10.1515/CCLM.2006.207.
3
Lead exposure is associated with decreased serum paraoxonase 1 (PON1) activity and genotypes.铅暴露与血清对氧磷酶1（PON1）活性降低及基因型有关。
Environ Health Perspect. 2006 Aug;114(8):1233-6. doi: 10.1289/ehp.9163.
4
5
Serum superoxide dismutase 3 (extracellular superoxide dismutase) activity is a sensitive indicator of Cu status in rats.血清超氧化物歧化酶3（细胞外超氧化物歧化酶）活性是大鼠铜状态的敏感指标。
J Nutr Biochem. 2005 Nov;16(11):682-92. doi: 10.1016/j.jnutbio.2005.03.009. Epub 2005 Aug 2.
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Ischemic heart disease as deficiency disease.缺血性心脏病作为营养缺乏病。
Cell Mol Biol (Noisy-le-grand). 2004 Dec;50(8):877-84.
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Vascular extracellular superoxide dismutase activity in patients with coronary artery disease: relation to endothelium-dependent vasodilation.
Circulation. 2000 May 16;101(19):2264-70. doi: 10.1161/01.cir.101.19.2264.
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Plasma extracellular superoxide dismutase levels in an Australian population with coronary artery disease.
Arterioscler Thromb Vasc Biol. 1998 Dec;18(12):1915-21. doi: 10.1161/01.atv.18.12.1915.
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Serum lipid peroxide level and blood superoxide dismutase activity in workers with occupational exposure to lead.
Int Arch Occup Environ Health. 1985;56(2):119-27. doi: 10.1007/BF00379383.
10
Red cell superoxide dismutase activity as an index of human copper nutrition.
J Nutr. 1985 Dec;115(12):1650-5. doi: 10.1093/jn/115.12.1650.
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